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glycoside/infarction

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[Clinico-electrocardiographic, morphological and biochemical manifestations of glycoside poisoning in patients with myocardial infarct].

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The clinical manifestations of glycoside toxicosis were compared with the character of myocardial ultrastructural changes and the electrolyte shifts outside the infarction zone. Accumulation of glycogen in the cytoplasm of the muscle cells with simultaneous gross destruction of the myocardial
Cattle encephalon glycoside and ignotin (CEGI) injection is a compound preparation formed by a combination of muscle extract from healthy rabbits and brain gangliosides from cattle, and it is generally used as a neuroprotectant in the treatment of central and peripheral nerve injuries. However,
OBJECTIVE To investigate the effect of cornel iridoid glycoside (CIG), an ingredient extracted from traditional Chinese herb Cornus offificinalis, on neurological function and infarct size in rats as measured by magnetic resonance imaging (MRI) after ischemic stroke. METHODS Sprague-Dawley rats were
BACKGROUND The aim of the study was to examine and confirm the cardioprotective effect and mechanism of phenylethanoid glycoside-rich extract of Cistanche deserticola (PhG-RE), a well-known natural antioxidant-based active constituents, against ischemia/reperfusion injury using both in vivo and in
OBJECTIVE To observe the effect of Cornus Officinalis total glycosides (COTG) and Cornus polysaccharides (CP) on myocardial mitochondria and expression levels of glycogen synthase kinase-3β (GSK-3β) of acute myocardial infarction (AMI) rats. METHODS The AMI rat model was established by ligating the
Our purpose is to study the effect of different component ratio of Astragalus Total Saponins (ATS) and Verbena Total Glycosides (VTG) on the cerebral infarction area and the serum biochemical indicators in the focal cerebral ischemia-reperfusion rat model. Compared with the model group, different
Following the guide of the publications written by Weissler, Thomas, Ahmed, Perry, Buyukoztur, Rahimotola, the Authors have executed on 25 patients with results of the myocardic infarction a polygraphic examination, comparing the variations of the intervals of the systolic time (STI) with the

[Ventricular arrhythmias in reduced glycoside tolerance and digitalis poisoning together with myocardial infarct].

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Electrophysiological fundamental principles of the heart muscle cell and the conduction system are discussed in detail. Basic differences are described. Pathophysiology of ventricular arrhythmias is emphasized, especially the ectopic conduction development and conduction defect of the reentry

Haemodynamic effects of prenaterol and cardiac glycosides in patients with recent myocardial infarction.

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Prenalterol was given to twenty five patients with acute myocardial infarction. Thirteen patients (group B) had signs of cardiac decompensation while twelve (group A) had not. Ejection fraction was determined with 99mTc-labelled red blood cells before and after prenalterol (50 micrograms kg-1 body

[Evaluation of bicycle ergometry before and after administration of cardiac glycosides to patients with history of myocardial infarct].

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[On the characteristic peculiarities of action of cardiac glycosides during combined therapy of myocardial infarct].

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[Arrhythmias during treatment of acute myocardial infarct by cardiac glycosides and possibilities of their prevention].

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[Concentrations of glycosides in the myocardium and other tissues following experimental myocardial infarct in the dog].

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[EFFECT OF CARDIAC GLYCOSIDES ON THE CORONARY CIRCULATION AND CARDIAC OXYGEN UPTAKE IN EXPERIMENTAL MYOCARDIAL INFARCTION].

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