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wood/necrosis

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A GUMMOSIS OF CITRUS ASSOCIATED WITH WOOD NECROSIS.

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Pulmonary responses after wood chip mulch exposure.

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Organic Dust Toxic Syndrome (ODTS) is a flu-like syndrome that can occur after inhalation of cotton, grain, wood chip dusts, or other organic dusts or aerosols. We investigated whether inflammatory pulmonary responses occur, even after relatively brief, low-level wood chip mulch exposure. Six
Multiple studies show that particulate mass (PM) generated from incomplete wood combustion may induce adverse health issues in humans. Previous findings have shown that also the PM from efficient wood combustion may induce enhanced production of reactive oxygen species (ROS), inflammation, and

Experimental acute yellow-wood (Terminalia oblongata) intoxication in sheep.

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An aqueous suspension of air-dried, hammer-milled leaf of Terminalia oblongata (yellow-wood) was administered to sheep by gavage, as a single dose of 5 to 20 g (dry weight)/kg body weight. Doses of 15 g/kg, or more, caused depression, inappetence, abdominal pain and reduced ruminal movements within

Quantitative Assessment of Grapevine Wood Colonization by the Dieback Fungus Eutypa lata.

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Eutypa lata is a fungal pathogen causing severe dieback in vineyards worldwide. This fungus colonizes vines through pruning wounds, eventually causing a brown sectorial necrosis in wood as well as stunted vegetative growth. Several years may pass between infection and the expression of external

Cultivar- and wood area-dependent metabolomic fingerprints of grapevine infected by Botryosphaeria dieback

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Botryosphaeria dieback is one of the most significant grapevine trunk diseases that affects the sustainability of the vineyards and provokes economic losses. The causal agents, Botryopshaeriaceae species, live in and colonize the wood of the perennial organs causing wood necrosis. Diseased vines

Pathogenesis of Frog Virus 3 ( Ranavirus, Iridoviridae) Infection in Wood Frogs ( Rana sylvatica).

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Wood frogs ( Rana sylvatica) are highly susceptible to infection with Frog virus 3 (FV3, Ranavirus, Iridoviridae), a cause of mass mortality in wild populations. To elucidate the pathogenesis of FV3 infection in wood frogs, 40 wild-caught adults were acclimated to captivity, inoculated orally with a

[Hepatoprotector properties of polyphenolic complexes from wood and cell culture of Maackia amurensis].

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Polyphenolic complexes (PPCs) extracted from core wood and cell culture of Maackia amurensis decrease the acute toxicity of tetrachloromethane, suppress the necrosis of hepatocytes and the cellular infiltration in liver parenchyma, prevent the development of fatty and protein dystrophy in the liver,

First Report of Wood Canker Caused by Neoscytalidium dimidiatum on Grapevine in California.

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In May 2012 in the Coachella valley, Riverside County, California, the decline of vines in table grape (Vitis vinifera) vineyards was observed. Foliar symptoms consisted of shoot blight with wilting and necrosis of leaves and drying and shriveling of berries. In some cases, the entire vine collapsed

Chemical composition and anti-inflammatory activity of Chamaecyparis obtusa f.formosana wood essential oil from Taiwan.

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Chamaecyparis obtusa Siebold & Zucc. f.formosana Hayata (Cupressaceae) is one of the treasured conifers in Taiwan. In this study, the wood essential oil of the species was extracted and its anti-inflammatory bioactivities analyzed. The composition analysis of the essential oil yielded a total of 57

Toxicity and bioaccumulation of the wood preservative copper dimethyldithiocarbamate in tissues of Long-Evans rats.

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This study examined the toxicity and accumulation of copper in the livers and kidneys of Long-Evans rats after a subacute exposure to copper dimethyldithiocarbamate (CDCC) wood preservative. CDDC was recently introduced as an alternative to chromated copper arsenate (CCA) preserved wood. Female rats

Nephrotoxicity effects of the wood preservative chromium copper arsenate on mice: histopathological and quantitative approaches.

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Chromium copper arsenate (CCA) was used for the protection of wood building materials until the restriction by EPA in 2002. During a short period of time 14-24h, a comparative nephrotoxicity study was performed regarding the effects of CCA and its compounds per se. Histopathological and

Absence of proinflammatory cytokine gene expression in nasal biopsies from wood surface-coating industry workers.

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Symptoms of nasal, pharyngeal and ocular discomfort have been reported among workers in the wood surface-coating industry. Symptoms were reported more often by workers using ultraviolet radiation-curable acrylate coatings (UV coatings), which contain potential chemical sensitizers, than by those

[Wood smoke condensate had weak proliferation and strong necrotic effects on human airway smooth muscle cells].

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OBJECTIVE To investigate the effect of wood smoke condensate (WSC) on proliferation and necrosis of human airway smooth muscle cells (HASMCs). METHODS Primary cultured HASMCs between passage 2 and 8 were divided into 3 groups: a control group, a WSC group and a cigarette smoke condensate (CSC)

Phenolic Responses to Esca-Associated Fungi in Differently Decayed Grapevine Woods from Different Trunk Parts of 'Cabernet Sauvignon'.

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Alterations in phenolic contents were studied in Esca symptomatic (Sym) and asymptomatic (Asym) vines of 'Cabernet Sauvignon' based on wood conditions (healthy, HLT; necrotic, Nec; and rotten, Rot) and vine parts (head, trunk, and rootstock). In Asym vines, only Alternaria alternate was identified
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