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west nile fever/asthenia

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Asymmetric Weakness and West Nile Virus Infection.

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BACKGROUND Weakness is a common presentation in the emergency department (ED). Asymmetric weakness or weakness that appears not to follow an anatomical pattern is a less common occurrence. Acute flaccid paralysis with no signs of meningoencephalitis is one of the more uncommon presentations of West

Delayed-onset and recurrent limb weakness associated with West Nile virus infection.

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Human neurologic illness following infection with West Nile virus (WNV) may include meningitis, encephalitis, and acute flaccid paralysis (AFP). Most WNV-associated AFP is due to involvement of the spinal motor neurons producing an anterior (polio)myelitis. WNV poliomyelitis is typically

Unilateral faciobrachial weakness: An unusual neuromuscular presentation Of West Nile Virus infection.

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West Nile virus infection in 2002: morbidity and mortality among patients admitted to hospital in southcentral Ontario.

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BACKGROUND In August and September 2002 an outbreak of West Nile virus (WNV) infection occurred in southern Ontario. We encountered a number of seriously ill patients at our hospitals. In this article we document the clinical characteristics of these cases. METHODS We conducted a retrospective chart

West Nile virus infection among health-fair participants, Wyoming 2003: assessment of symptoms and risk factors.

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Wyoming experienced heavy West Nile virus (WNV) activity for the first time in 2003 and the area hardest hit was Goshen County. Little was known about the epidemiology of WNV in this region. This study describes the symptomatology of WNV and the association between certain behaviors and infection in

Asymmetric flaccid paralysis: a neuromuscular presentation of West Nile virus infection.

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The neuromuscular aspects of West Nile virus (WNV) infection have not been characterized in detail. We have studied a group of six patients with proven WNV infection. All cases presented with acute, severe, asymmetric, or monolimb weakness, with minimal or no sensory disturbance after a mild

Community-acquired West Nile virus infection in solid-organ transplant recipients.

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BACKGROUND West Nile virus (WNV) is rapidly spreading through North America. In the general population, the majority of WNV infections are asymptomatic. During 2002, an outbreak of WNV occurred in Toronto, Canada. We observed four cases of severe symptomatic community-acquired WNV infection in our

West Nile virus infection: a new acute paralytic illness.

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OBJECTIVE To determine the clinical, laboratory, electrodiagnostic, radiologic, and pathologic characteristics that define the spectrum of CNS disease caused by West Nile virus (WNV) infection. METHODS The records of all patients hospitalized at the Cleveland Clinic from August 2002 to September

Acute flaccid paralysis and West Nile virus infection.

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Acute weakness associated with West Nile virus (WNV) infection has previously been attributed to a peripheral demyelinating process (Guillain-Barré syndrome); however, the exact etiology of this acute flaccid paralysis has not been systematically assessed. To thoroughly describe the clinical,

Two case reports of neuroinvasive west nile virus infection in the critical care unit.

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We describe the clinical course of two cases of neuroinvasive West Nile Virus (WNV) infection in the critical care unit. The first case is a 70-year-old man who presented during summer with mental status changes. Cerebrospinal fluid (CSF) analysis revealed pleocytosis with lymphocyte predominance.

West Nile Virus Infection in the Immunocompromised Patient.

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West Nile virus infection has become the predominant cause of flavivirus-associated encephalitis in the US. While 80 % of infected individuals are asymptomatic, 20 % develop symptoms including fever, headache, transient rash and gastrointestinal symptoms. Among the immunocompetent population, 1 in

Neuromuscular Manifestations of Human West Nile Virus Infection.

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Physicians in areas with active West Nile virus (WNV) transmission should be aware that WNV infection can present as a polio-like syndrome and that the spectrum of neuromuscular signs and symptoms may range from acute flaccid paralysis in the absence of fever or meningoencephalitis to subjective

Electrodiagnostic features of acute paralytic poliomyelitis associated with West Nile virus infection.

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West Nile virus (WNV) infection is a potentially fatal disease, with meningoencephalitis being its most common neurological manifestation. Guillain-Barré syndrome (GBS) has also been described, but acute paralytic poliomyelitis has only recently been recognized. We report the clinical and

Recovery and prognosticators of paralysis in West Nile virus infection.

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Previous studies have demonstrated that lesions of the anterior horn motor neurons are the primary pathology in patients with paralysis due to West Nile virus (WNV) infection. To characterize recovery and identify prognostic factors for the recovery of paralysis, we investigated 11 patients with

Clinical West Nile virus infection in 2 horses in western Canada.

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Two horses had a history of ataxia and weakness or recumbency. One recovered and was diagnosed with West Nile virus (WNV) infection by serologic testing. The other was euthanized; it had meningoencephalomyelitis, WNV was detected by polymerase chain reaction. West Nile virus infection is an emerging
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