aspartate amino transferase/infarction

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Effect of administration of carnitine on the severity of myocardial infarction induced by isoproterenol in rats.

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The effect of administration of carnitine on the severity of myocardial infarction in rats induced by isoproterenol was studied by following histopathological and biochemical parameters. Carnitine afforded partial protection against myocardial infarction. Serum aspartate amino transferase (GOT) and

Comparison of troponin T and enzyme levels in acute myocardial infarction and skeletal muscle injury.

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BACKGROUND The objective of this study was to compare the levels of troponin T and enzymes levels in myocardial infarction and skeletal muscle injury. METHODS This study was carried out at Basic Medical Sciences Institute, JPMC Karachi, Pakistan. Ninety subjects were selected. Thirty controls,

Relationship between different enzymatic estimations of the infarct size.

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In 592 patients with an acute myocardial infarction different enzymatic estimations of the infarct size were compared. Heat stable lactate dehydrogenase (LD) was analysed every 12 hours for 48-108 hours. Aspartate amino transferase (ASAT) was analysed once daily for 3 days. In a subgroup creatine

Michaelis-Menten constant of serum aspartate amino transferase (AST) in heart and liver diseases.

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The activity and the Km (Michaelis-Menten constant) of serum aspartate amino transferase (AST) were investigated in normal subjects, in patients with recent myocardial infarction, and in patients with infectious hepatitis. AST activity was elevated in both groups of patients. While the Km value of

Effect of saturated, omega-3 and omega-6 polyunsaturated fatty acids on myocardial infarction.

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Dietary fatty acids have cholesterol lowering, antiatherogenic, and antiarrhythmic properties that decrease the risk of myocardial infarction (MI). This study was designed to study the effects of various oils rich in either polyunsaturated (omega-3 or omega-6) fatty acids (PUFA) or saturated fatty

Hyponatraemia and severity and outcome of myocardial infarction.

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A total of 235 consecutive patients admitted to a coronary care unit were investigated for serum electrolyte and urea concentrations; activities of aspartate amino-transferase, lactate dehydrogenase, and lactate dehydrogenase isoenzymes; electrocardiographic changes; clinical state; and outcome.

[Biological diagnosis of myocardial infarction].

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The biochemical diagnosis of myocardial infarction is mainly based on assays of serum enzymes. The most commonly used markers are creatine kinase, lactic dehydrogenase and aspartate amino-transferase. They are useful to diagnose the infarction, determine its time of onset and evaluate its

"Spontaneous" platelet aggregation in whole blood in diabetic and non diabetic survivors of acute myocardial infarction.

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There is increasing evidence that platelet thrombi play an important role in the pathogenesis of acute myocardial infarction (AMI). We compared "spontaneous" platelet aggregation in whole blood in 17 non-diabetic and 12 diabetic subjects on admission with AMI. There was no significant difference in

Effect of alpha-tocopherol on isoproterenol induced myocardial infarction in rats--electrocardiographic biochemical and histological evidences.

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The effect of alpha-tocopherol (6 mg/100 g body wt, orally, daily for 90 days) pretreatment in isoproterenol (20 mg/100 g body wt, subcutaneously, twice at an interval of two days at the end of the alpha-tocopherol pretreatment) induced myocardial infarction was studied in rats. Isoproterenol

Effect of aspartate and glutamate on experimental myocardial infarction in rats.

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Cardiac necrosis was produced in rats by administering isoproterenol sulphate (85 mg/kg, sc for 4 days). The myocardial damage was proved by observing the elevated levels of serum aspartate amino-transferase, lactate dehydrogenase and creatine phosphokinase and the changes were confirmed by

[Enzymatic activity of the cerebrospinal fluid in patients with cerebral infarct].

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Dynamic measurements in the cerebrospinal fluid (CSF) of the activities of lactate dehydrogenase (LDH), its first isoenzyme (LDH1), aspartate amino transferase (AsAT), alanine amino transferase (AlAT), creatine phosphokinase (CPK), gamma-glutamyl transpeptidase (GGT) and alkaline phosphatases (ALP)

Cardiotoxicity and myocardial infarction-associated DNA damage induced by thiamethoxam in vitro and in vivo: Protective role of Trigonella foenum-graecum seed-derived polysaccharide.

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The risk of pesticides on the human health and environment has drawn increasing attention. Today, new tools are developed to reduce pesticide adverse effects. This study aimed to evaluate the toxicity induced by, thiamethoxam (TMX), and the cytoprotective effect of a novel polysaccharide, named

Post partum creatine phosphokinase and its muscle-brain isoenzyme elevation and transient Q-wave in a patient with idiopathic hypertrophic subaortic stenosis.

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A primigravida with idiopathic hypertrophic subaortic stenosis, New York Heart Association Classification III, developed acute chest pain with significant ST segment depression together with a new Q-wave in chest lead V6 on the electrocardiograph following delivery under lumbar epidural analgesia.

Is continuous warm retrograde blood cardioplegia completely safe for coronary artery surgery?

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Sixty consecutive coronary patients operated on by the same team in 1992 were divided into two groups: group 1 (30 patients) using intermittent oxygenated cold Fresenius solution antegrade and retrograde (FR), group 2 (30 patients) using warm retrograde blood cardioplegia (WRC) with the Fremes

Elevation of serum creatine phosphokinase and its MB isoenzyme during normal labor and early puerperium.

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BACKGROUND Chest pain or discomfort are infrequent complaints among women during labor and early puerperium, but when present they raise the suspicion of myocardial ischemia. The diagnosis of the latter is based upon serum elevatIon of certain enzymes, such as aspartate amino transferase, lactate

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