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cholecystitis/protease

Nuoroda įrašoma į mainų sritį
StraipsniaiKlinikiniai tyrimaiPatentai
12 rezultatus

Leukocytic proteases in gallbladder pathology. Experimental acute cholecystitis in dogs.

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Prisijungti Registracija
OBJECTIVE Preclinically, HIV protease inhibitors radiosensitize tumors with activated PI3-kinase/Akt pathway. We determined the toxicity of nelfinavir chemoradiotherapy in borderline resectable and unresectable pancreatic cancer. METHODS Oral nelfinavir (2 x 1,250 mg) was started 3 days before and

Agonists of protease-activated receptors 1 and 2 stimulate electrolyte secretion from mouse gallbladder.

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Cholecystitis is one of the most common gastrointestinal diseases. Inflammation induces the activation of proteases that can signal to cells by cleaving protease-activated receptors (PARs) to induce hemostasis, inflammation, pain, and repair. However, the distribution of PARs in the gallbladder is

[Dynamics of the total proteolytic activity and kinin system components of the blood in acute cholecystitis].

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Under study was the dynamics of changes of the activity of components of the kinin system and proteolytic activity of blood in 32 patients with acute cholecystitis. The results have shown that cholecystic patients and patients with the diseases of biliary ducts, especially in the exacerbation stage,

[Post-traumatic cholecystitis].

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Post-traumatic cholecystitis occurs mostly in young males 8-16 days after severe injury. Gallstones are of no importance in its pathogenesis. Circulatory shock causes severe damage to the small bowel and the liver, particles of the destroyed cells becoming endogenous toxins. Presumably an increased
Treatment options for HIV-infected patients can be limited due to viral drug resistance to antiretroviral agents. Enfuvirtide (ENF) is an injectable entry/fusion inhibitor that is effective in achieving viral suppression when used in combination with protease inhibitors (PIs) in patients with

Cytochemical investigation of neutral proteases in polymorphonuclear (PMN) neutrophils in acute inflammatory diseases.

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Neutral proteases can be released from PMN neutrophils in blood smears from healthy subjects by incubation with NaCl-borate buffer. The activity of the PMN proteases can be revealed by the degradation of erythrocytes and plasma within ring-shaped areas centered around each neutrophil (halo effect).

[The use of regional lymphotropic therapy in patients with acute complicated cholecystitis].

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The results of the regional lymphotropic therapy application in 58 patients with an acute complicated cholecystitis are presented. The drop-infusion administration of the lymph outflow promoting drugs, antibiotics and antiseptics, proteases inhibitors and cytostatics, immunostimulants was conducted

Is ritonavir-boosted atazanavir a risk for cholelithiasis compared to other protease inhibitors?

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OBJECTIVE To compare the incidence of complicated cholelithiasis in patients receiving ritonavir-boosted atazanavir (ATV/r)- containing antiretroviral therapy with those on other protease inhibitors (PIs). METHODS We conducted a single-center retrospective cohort study of patients who started either

Localization of Tubulin from the Carcinogenic Human Liver Fluke, Opisthorchis viverrini.

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BACKGROUND Opisthorchiasis caused by Opisthorchis viverrini, is of considerable public health importance in Southeast Asia, particularly in Lao People's Democratic Republic and Thailand. The infection is associated with a number ofhepatobiliary diseases including cholangitis, obstructive jaundice,
Tritrichomonas species flagellates (IMC strain) were isolated from the biliary tract of an individual who had developed cholecystitis as a complication of acquired agammaglobulinemia. Sequence analysis of Tritrichomonas sp. (IMC clone 2 (cl2)) was performed for several genetic regions including the

Effects of bioactive agents on biliary motor function.

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Our understanding of biliary motility under normal and pathophysiologic conditions is still incomplete, but there have been recent advances. Of particular interest are the mechanisms involved in gallbladder filling and emptying, with a focus on understanding the processes underlying impaired
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