ionol/infarction

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StraipsniaiKlinikiniai tyrimaiPatentai

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[Prevention of a disorder in the electrical stability of the heart in experimental myocardial infarct by using preliminary adaptation to brief stress exposures and the antioxidant ionol].

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Prisijungti Registracija

Preliminary adaptation to short-term stress was shown to prevent the fall of the heart fibrillation threshold and its increased ectopic activity which is usually observed in experimental myocardial infarction. This protective effect involves an enhanced activity of the organism's antioxidant

[Elimination of disorders of electric stability of the heart during post-infarct cardiosclerosis using adaptation to short-term stress and the antioxidant ionol].

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Prisijungti Registracija

The experiments were carried out on male Wistar rats (300-400 g) subject to open chest surgery under nembutal anesthesia. One group of rats with postinfarction cardiosclerosis (PC) was exposed to short-term immobilization stress for 15 days one month after the occlusion of the descending branch of

[Effect of dibunol on lipid peroxidation and alpha-tocopherol levels in the rat liver in myocardial infarction].

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Prisijungti Registracija

Lipid peroxidation in rat liver tissue homogenate was studied by evaluation of diene conjugates and Schiff bases content within 24 hrs after myocardium infarction. Tocopherol was estimated using spectrofluorimetric procedure. Ionol was administered intraperitoneally at a dose of 120 mg/kg within 15

[Combined use of hyperbaric oxygenation and antioxidants in the therapy of experimental myocardial infarct].

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Prisijungti Registracija

In rabbits with experimental myocardial infarction, the combined use of hyperbaric oxygenation and ionol greatly potentiated the effect of the antioxidant on contractile function of the left ventricle, the time course of antioxidant lipid activity and superoxide dismutase activity of the myocardium.

Stress damage to nonischemic divisions of the heart in experimental infarction and its prevention.

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Prisijungti Registracija

Recently, it has been shown that nonischemic parts of the heart in myocardial infarction were separated from ischemic damaged ones by a sharp border zone. In this connection, the disturbance of contractile function of the myocardium of nonischemic parts is suggested to result from the

[The prevention of a postinfarct fall in pressure and hyperactivation of the aortic endothelium in the rat by using the antioxidant ionol].

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Prisijungti Registracija

The stress concomitant with experimental myocardial infarction resulted in endothelial hyperactivation of the isolated rat aorta. The hyperactivation was manifested in potentiated inhibition of norepinephrine-induced contractions of vascular smooth muscle, increased endothelium-dependent relaxation

[Limited area of coronary-occlusion myocardial infarct in rats undergoing antioxidant therapy].

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Prisijungti Registracija

The synthetic antioxidant dibunol, (ionol. 2,6-ditret-butyl-4-methylphenol) produces the limitation of the zone of the coronaro-occlusion myocardial infarction in rats by 15.8 and 24.2% on day 7 during daily oral administration in doses of 80 and 120 mg/kg, respectively. In the doses used, dibunol

[Limitation of ischemic necrosis by the use of antioxidant ionol].

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Prisijungti Registracija

Inhibitory effect of Ionol on lipid peroxidation (LPO) in the infarction zone and out of it after experimental myocardial infarction in the experiments on rats was investigated. The results of measurements, performed by two independent methods: point counting and the computer image analysis were

[DNA changes in a nonischemic area of the rat myocardium in experimental myocardial infarct and its prevention].

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Prisijungti Registracija

DNA damage and repair in the nonischemic area of the heart were analyzed after experimental myocardial infarction to evaluate the effect on these processes of the beta-blocker inderal and lipid peroxidation inhibitor ionol. It was found that in the nonischemic area of the heart, DNA damage was

[Possible role of lipid peroxidation in the pathogenesis of arrhythmias in myocardial infarct].

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Prisijungti Registracija

The effect of myocardial infarction sustained by rats on the resistance of their isolated auricles to H2O2, an inductor of lipid peroxidation (LP), was studied. Atrial resistance to the LP inductor depends on the level of developed tension (DT) and the decrease of DT leads to augmentation of atrial

Adaptation to short-term stress prevents post-infarction hyperactivation of the endothelium and decrease in blood pressure in rats.

Straipsnius versti gali tik registruoti vartotojai

Prisijungti Registracija

The aim of the study was to elucidate the possibility of preventing the decrease in blood pressure (BP) and the endothelial hyperactivation that are induced by experimental myocardial infarction in rats. The endothelial hyperactivation manifested itself in potentiated endothelium-dependent

[Disorders of contractile function and adrenoreactivity of the portal vein in emotionally-painful stress and experimental myocardial infarct and their prevention by means of membrane protectors].

Straipsnius versti gali tik registruoti vartotojai

Prisijungti Registracija

The effect of emotional-painful stress (EPS) and experimental myocardial infarction on portal vein contractile function, adrenoreactivity , and resistance to the glucose shortage was studied on the rat. The myocardial infarction was shown to result in shifts of the smooth muscle contractile function

Disturbance of contractile function of vena portae in stress and infarction and its prevention.

Straipsnius versti gali tik registruoti vartotojai

Prisijungti Registracija

Emotional painful stress was shown to result in a significant depression of the portal vein contractile function accompanied by its decreased adrenoreactivity and its increased dependence on changes of external temperature, calcium and glucose. Experimental myocardial infarction caused similar

[Lipid peroxidation in the myocardium in experimental infarct].

Straipsnius versti gali tik registruoti vartotojai

Prisijungti Registracija

Changes in the content of lipid peroxidation (LP) products and activities of antioxidant enzymes--superoxide dismutase, glutathione peroxidase and catalase in myocardium of rats after experimental infarction as well as after pretreatment with antioxidant ionol, beta-adrenoblocker inderal and

[Effect of adaptation to short-term stress exposure on the fibrillation threshold and ectopic activity of the heart in experimental myocardial infarct].

Straipsnius versti gali tik registruoti vartotojai

Prisijungti Registracija

Preliminary adaptation to short-term stress was shown to prevent the decrease in the heart fibrillation threshold and an increase in ectopic activity which is usually observed in experimental myocardial infarction. This protective effect involves an enhanced activity of the antioxidant system.

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