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rickets/paprastasis kukurūzas

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StraipsniaiKlinikiniai tyrimaiPatentai
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Dietary level of fat, calcium, and vitamins A and D3 as contributory factors to rickets in poults.

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Two experiments were conducted on dietary predisposition to rickets in poults. The first experiment compared fat type (corn oil or tallow), level of added fat (3.5 or 7%), vitamin D3 (900 or 2,400 IU/kg feed), and total calcium (.6, 1.2, or 3%) inclusion in the diet. Poults fed diets supplemented

Spatial distribution of calcium in food, water and soil and its possible influence on rickets disease in Northern Nigeria.

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Since the 1990s, children of the Gbagyi tribe in Northern Nigeria have been suffering severe rickets with an incidence of up to 40% in the children's generation. The disease seems to be prevalent in an area of approximately 100 km(2) south-east of Kaduna. According to broad medical studies in that

Interrelationships of vitamin D, bone metabolism and blood calcium concentration in the chick.

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It is not known if the effects of vitamin D deficiency on chick bone are due to direct actions of the vitamin or if they are secondary to other changes, such as hypocalcemia. Day-old cockerels were fed either a rachitogenic diet containing no Ca (-D-Ca), 1.4% Ca (-D), or 3% Ca (-DHiCa) and given

Excess dietary vitamin A in the growing chick: effect of fat source and vitamin D.

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Studies were conducted to determine the nutritional interrelationship between different fat sources, vitamin D3 (vit D) and excess dietary vitamin A (vit A) in the growing chick. Birds tolerated as much as 30 times the recommended level of vit A without compromising performance or skeletal
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