strychnine/dantų ėduonis

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Responses of cultured rat trigeminal ganglion neurons to bitter tastants.

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The initial steps in taste and olfaction result from the activation by chemical stimuli of taste receptor cells (TRCs) and olfactory receptor neurons (ORNs). In parallel with these two pathways is the chemosensitive trigeminal pathway whose neurons terminate in the oral and nasal cavities and which

Effect of trigeminal tractotomy on behavioral response to dental pulp stimulation in the monkey.

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Trigeminal tractotomy near the level of the obex was carried out in 10 macaque monkeys. Behavioral responses were evaluated by a quantitative paradigm measuring lever-press responses to electrical stimulation of the dental pulp or facial skin, and by assessing facial response to cutaneous

Glutamate activates neuronal discharges and rhythmical jaw movements in the rat.

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Rhythmical jaw movements (RJM) were induced by stimulation of the cerebral peduncle or the oral cavity in the anesthetized rat and iontophoretic application of glutamate was examined on each reticular neuron with phase-dependent rhythmical activity during RJM (RJM-phasic neuron). Glutamate activated

Relief of cancer pain by glycine transporter inhibitors.

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BACKGROUND Recent studies have revealed the antinociceptive effects of glycine transporter (GlyT) inhibitors in neuropathic pain models such as sciatic nerve-injured and diabetic animals. Bone cancer can cause the most severe pain according to complex mechanisms in which a neuropathic element is

Practical toxicologic diagnosis.

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Strychnine toxicosis is characterized by inducible tetanic seizures and metaldehyde poisoning by fine fasciculations progressing to generalized tremors and seizures. Intoxication with 1080 causes seizures, random running movements, vomiting, defecation, urination, acidosis and hyperglycemia.

Characterization of two mutations, M287L and Q266I, in the α1 glycine receptor subunit that modify sensitivity to alcohols.

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Glycine receptors (GlyRs) are inhibitory ligand-gated ion channels. Ethanol potentiates glycine activation of the GlyR, and putative binding sites for alcohol are located in the transmembrane (TM) domains between and within subunits. To alter alcohol sensitivity of GlyR, we introduced two mutations

The action of ergotamine on the intracranial venous pressure and on the cerebral venous outflow of the dog.

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The effect of ergotamine and dihydroergotamine on the cerebral circulation was studied in the dog, anaesthetized with chloralose, by recording the intracranial venous pressure and the venous outflow from the superior cerebral vein. Under optimal experimental conditions, ergotamine (5 to 10 mug./kg.)

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