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Puslapis 1 nuo 118 rezultatus
The kinetic dissection of transport from metabolic trapping during substrate uptake by intact cells. Uridine uptake by quiescent and serum-activated Nil 8 hamster cells and their murine sarcoma virus-transformed counterparts.
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1. We present a theoretical analysis of the tandem processes of transport and metabolic trapping which together constitute uptake of a substrate by intact cells. 2. Transport is assumed to occur by means of a simple carrier here analysed in its general form. Trapping is assumed to occur by a simple
[Kinetics of the development of Rous sarcoma virus (Schmidt-Ruppin strain). Incorporation of uridine and arginine in associated intracytoplasmic particles. Study with high resolution autoradiography].
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[Uridine-cytidine kinase isoenzymes of rat sarcoma M-1].
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Inhibition of uridine transport through sarcoma-180 cell membrane by anthracycline antibiotics.
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Rous sarcoma virus-induced changes in uridine and UMP metabolism in chick chorioallantoic membrane.
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[Method of preparation of 2-C14-cytidine and uridine nucleotides with the use of enzyme preparation from M-1 rat sarcoma].
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Uptake of uridine and its incorporation in RNA of Sarcoma 180 ascites tumour cells following in vivo gamma radiation and mitomycin C treatment.
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Induction of deoxycytidylate deaminase and uridine kinase and activation of cellular DNA synthesis in the course of transformation of chicken embryo cells infected by Rous sarcoma virus in vitro.
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The effect of D-glucosamine on the adenine and uridine nucleotides of sarcoma 180 ascites tumor cells.
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Rapid transformation of cells by Rous sarcoma virus.
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The conditions for synchronous transformation of chick embryo cells by infection with Rous sarcoma virus are studied. Two factors, the treatment of cells with DEAE-dextran and the use of cells which grow rapidly following virus infection, are found to be most important. Under the conditions
Establishment of simian sarcoma virus, type 1 (SSV-1)-transformed non-producer marmoset cell lines.
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Simian sarcoma virus, type 1 (SSV-1)-transformed non-producer cell lines were established by infection of normal marmoset fibroblast cells (HF) with limiting dilutions of SSV-1. Four focus-derived cell lines were identified as non-producers by assay of culture fluids for focus-forming activity and
Noncoding region between the env and src genes of Rous sarcoma virus influences splicing efficiency at the src gene 3' splice site.
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Viral RNA and proteins in chicken embryo fibroblasts infected with different cloned variants of the Prague strain Rous sarcoma virus (RSV) were analyzed. The ratio of immunoprecipitated pp60src to the gag gene product p27 in Prague A (PrA) and Prague B (PrB) RSV-infected cells was two to three times
Biochemical and cytotoxic actions of 3,6-dihydroxy-4,5-dimethylphthalaldehyde in sarcoma 180 cells.
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The replication of Sarcoma 180 cells in culture was inhibited by 3,6-dihydroxy-4,5-dimethylphthalaldehyde (HMPA). The inhibition of growth caused by HMPA was evident after treatment of cells with drug for only 15 min. This exposure period caused decreased in (a) cloning efficiency, (b) transport
DNA synthesis inpermeable mouse ascites sarcoma cells.
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DNA synthesis was studied in mouse ascites sarcoma cells using a permeable cell system. The sarcoma was induced by the Schmidt-Ruppin strain of Rous sarcoma virus. The cells were made permeable to nucleoside triphosphates by treatment with a hypotonic buffer containing 10 mM Tris Cl, 4 mM MgCl2, 1
Uridine diphosphate reductase of Ehrlich ascites tumor is insensitive to hydroxyurea.
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Uridine diphosphate (UDP) reductase was isolated in the supernatant fraction obtained after the acidification of the cytosol of Ehrlich ascites tumor cells, and was found insensitive to 10 mM hydroxyurea. However, cytidine diphosphate (CDP) reductase, being separated concurrently in the precipitate
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