xanthurenic acid/cukrinis diabetas

Gestational diabetes mellitus (GDM) is a form of diabetes that is first recognised during pregnancy, with no evidence of pre-existing type 1 or type 2 diabetes. The prevalence of GDM has been rising steadily over the past few decades, coinciding with the ongoing epidemic of obesity and type 2

Studies on the urinary excretion of xanthurenic acid in diabetics.

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Xanthurenic acid (XA), kynurenic acid (KA) and creatinine in fasting urine were determined by reversed phase high pressure liquid chromatography in order to investigate the distortion of tryptophan metabolites in diabetes mellitus. The results of ten patients with non-insulin dependent diabetes

Concentrations of various tryptophan metabolites are higher in patients with diabetes mellitus than in healthy aged male adults.

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Tryptophan metabolites in plasma samples from 20 male subjects with type 2 diabetes mellitus (T2DM) and 20 nondiabetic reference males were analyzed by ultra high performance liquid chromatography. Tryptophan levels in the diabetic subjects were significantly lower than those in nondiabetic

The role of kynurenines in diabetes mellitus.

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Although different forms of diabetes are known to exist, there are a number of factors which occur in these states all of which would contribute to an increase in the synthesis of the kynurenine metabolites of tryptophan, xanthurenic acid in particular. Conditions giving rise to increased kynurenine

[Effectiveness of the use of vitamin-enriched food premixes in the nutrition of children with insulin-dependent diabetes].

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Vitamin-deficient children suffering from diabetes mellitus received milk dishes and drinks enriched with vitamin-rich premixes produced in Austria (Roche) for 2 weeks. The diet efficacy was evaluated by the values of thiamine, riboflavin, 4-pyridoxic acid, 1-methylnicotinamide and xanthurenic acid

Kynurenine, Tetrahydrobiopterin, and Cytokine Inflammatory Biomarkers in Individuals Affected by Diabetic Neuropathic Pain

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Neuropathic pain is a common complication of diabetes with high morbidity and poor treatment outcomes. Accumulating evidence suggests the immune system is involved in the development of diabetic neuropathy, whilst neuro-immune interactions involving the kynurenine (KYN) and tetrahydrobiopterin (BH4)

Metabolomics identifies novel Hnf1alpha-dependent physiological pathways in vivo.

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Mutations in the HNF1A gene cause maturity-onset diabetes of the young type 3, one of the most common genetic causes of non-insulin-dependent (type 2) diabetes mellitus. Although the whole-body Hnf1a-null mouse recapitulates the low insulin levels and high blood glucose observed in human

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