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esophageal and gastric varices/protease
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6 resultaten
Preservation of platelet function in patients with cirrhosis and thrombocytopenia undergoing esophageal variceal ligation.
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Portal vein thrombosis in a patient with HIV treated with a protease inhibitor-containing regimen.
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We report a case of an HIV seropositive female patient treated with a protease inhibitor-containing regimen who developed recurrent severe life-threathening episodes of haematemesis over time, caused by ruptured oesophageal varices as a consequence of a portal vein thrombosis. Coagulation tests
Increased serum activity of matrix metalloproteinase-9 in patients with acute variceal bleeding.
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OBJECTIVE
Matrix metalloproteinases (MMP)-2 and -9 can degrade essential components of vascular integrity. The aim of this study was to investigate the association between those MMPs and variceal bleeding (VB).
METHODS
Fifteen controls, 12 patients with acute ulcer bleeding (UB) group, 37 patients
Changes in wound healing factors in liver cirrhosis after esophageal transection for esophageal varices.
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Quantitative changes in plasma wound healing factors in cirrhotic patients after esophageal transection were evaluated and compared with those of non-cirrhotic esophageal cancer patients (controls) after esophageal resection. Serum total protein, albumin and fibronectin were maintained at the same
[Progress in interventional radiology (IVR) in emergency medicine].
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IVR has attracted much attention in Japan over the past decade, and it is expected to be rapidly developed and widely used. Not surprisingly, IVR has already become essential in emergency medicine. This paper addresses IVR of the thoracic and abdominal areas and the pelvis in emergency medicine, in
Association of noncirrhotic portal hypertension in HIV-infected persons and antiretroviral therapy with didanosine: a nested case-control study.
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BACKGROUND
Noncirrhotic portal hypertension (NCPH) is a newly described life-threatening liver disease of unknown cause in human immunodeficiency virus (HIV)-infected persons. Postulated pathogenesis includes prolonged exposure to antiretroviral therapy, particularly didanosine.
METHODS
We performed
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