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hemosiderosis/kalium
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Fanconi syndrome and CKD in a patient with paroxysmal nocturnal hemoglobinuria and hemosiderosis.
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Fanconi syndrome and chronic kidney disease associated with paroxysmal nocturnal hemoglobinuria is rarely reported. We describe a 51-year-old woman with glomerular filtration rate decrease and hypokalemia, glucosuria, and proteinuria during a 4-year period. Paroxysmal nocturnal hemoglobinuria was
Renal Hemosiderosis among Iranian Transfusion Dependent β-Thalassemia Major Patients.
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Background: In recent years, the success in management of thalassemic patients, has allowed for some previously unrecognized complications including renal abnormalities to emerge. This prospective study aimed to investigate kidney iron overload by means of MRI T2* and also renal function based on
Heterogeneous phenotype of Hereditary Xerocytosis in association with PIEZO1 variants.
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Hereditary Xerocytosis (HX) is an autosomal dominantly inherited congenital hemolytic anemia associated with erythrocyte dehydration due to decreased intracellular potassium content resulting in increased mean corpuscular hemoglobin concentration. The affected members of HX families show compensated
Prey, parasites and pathology associated with the mortality of a juvenile gray whale (Eschrichtius robustus) stranded along the northern California coast.
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An eastern Pacific gray whale (Eschrichtius robustus) stranded off Pelican Point, Tomales Bay, California, USA, was examined for physiological parameters, prey, parasites and associated pathology. The whale was emaciated, and hematological examination revealed an elevation in hematocrit, serum
Subchronic toxicity of 3-chloro-4-(dichloromethyl)-5-hydroxy-2(5H)-furanone (MX) in Wistar rats.
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The subchronic (14-18 wk) toxicity of 3-chloro-4-(dichloromethyl)-5-hydroxy-2(5H)-furanone (MX), a mutagenic by-product in chlorinated drinking water, was evaluated in Wistar rats. In a range-finding study, MX was administered daily for 14 days by gavage in deionized water to male rats (five animals
[Biochemical, electrocardiographic and pathomorphological studies the monensin poisoning of sheep].
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Studies were carried out on the changes in the electrocardiogram, the content of sodium and potassium in the plasma and erythrocytes, and the morphology of the viscera of sheep spontaneously intoxicated with monensin. It was found that in the initial phases of poisoning the plasma level of Na and K
Renal function in children with beta-thalassemia major and thalassemia intermedia.
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In beta-thalassemia, profound anemia and severe hemosiderosis cause functional and physiological abnormalities in various organ systems. In recent years, there have been few published studies demonstrating proteinuria, aminoaciduria, low urine osmolality, and excess secretion of the tubular damage
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