hyperparathyroidism/carbohydrate

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[Diabetes mellitus and carbohydrate metabolism in primary hyperparathyroidism].

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It is generally known that patients with primary hyperparathyroidism (pHPT) feature disturbances in carbohydrate metabolism and hypertension. The incidence and prevalence of frank diabetes mellitus is significantly increased in these patients. The etiology and pathogenesis of the vascular and

Hyperparathyroidism and coexisting diabetes mellitus. Altered carbohydrate metabolism.

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Hyperparathyroidism was diagnosed in a 67-year-old diabetic man treated for 20 years with isophane insulin suspension, 40 to 45 units/day. It was also diagnosed in a 64-year-old diabetic with severe retinopathy and vascular disease, who was not dependent on insulin. In the first case, removal of a

Carbohydrates in the non-dialysable fractions of filtered urine in hyperparathyroidism.

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Diabetes mellitus with hyperparathyroidism: another indication for parathyroidectomy?

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BACKGROUND Patients with hyperparathyroidism have alterations in carbohydrate metabolism characterized by insulin resistance, hyperinsulinemia, and glucose intolerance. The clinical significance of these findings in the management of patients with diabetes mellitus (DM) after parathyroidectomy for

Peripheral insulin resistance in primary hyperparathyroidism.

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Carbohydrate metabolism was investigated in 9 patients with symptomatic primary hyperparathyroidism. Before and after parathyroidectomy intravenous and oral glucose tolerance test, tolbutamide test, arginine infusion test and insulin tolerance test were performed. During intravenous and oral glucose

Primary hyperparathyroidism is associated with decreased insulin receptor binding and glucose intolerance.

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We studied insulin receptor-binding and carbohydrate and metabolism in 15 patients with symptomatic primary hyperparathyroidism in comparison with 20 healthy controls. Insulin binding to monocytes and erythrocytes was measured by radioreceptor-ligand-assay. Furthermore, patients and controls were

Growth hormone and insulin-like growth factor binding protein-1 responses to oral glucose in patients with primary hyperparathyroidism.

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BACKGROUND GH and IGFBP-1 both play a role in glucose homeostasis. OBJECTIVE To assess the GH and IGFBP-1 responses to an oral glucose load and their relationship with glucose homeostasis in patients with primary hyperparathyroidism. METHODS A cross-sectional study with a control group followed by a

Carbohydrate metabolism in uraemia.

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OBJECTIVE Most uraemic patients are insulin resistant. This review focuses on the occurrence, mechanisms and consequences of this insulin resistance. Hypoglycaemia is also possible in a minority of uraemic patients; its causes are discussed at the end of the review. RESULTS Insulin resistance is

[Role of calcium in the metabolism of carbohydrates].

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The role of calcium in the metabolism of carbohydrates is rather complex. It has been shown particularly "in vitro" but also "in vivo", to act on beta cells and peripheral effectors. Cytosolic calcium is determinant for insulin secretion in beta cells. It may be increased by a transfer of

[Renal lithiasis in idiopathic hypercalciuria and primary hyperparathyroidism].

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The high incidence of renal lithiasis in hyperparathyroidism (55 p. 100) suggests that PTH plays a causal role in stone production. It also motivates a systematic search for primary hyperparathyroidism in all patients with renal stones although it is only found in about 7 p. 100 of cases. PTH acts

Evaluation of glucose tolerance, insulin secretion, and insulin action in patients with primary hyperparathyroidism before and after surgery.

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Glucose tolerance, insulin secretion, and insulin sensitivity were evaluated in 8 asymptomatic patients with primary hyperparathyroidism (PHPT) before and at least 8 weeks after surgical correction of PHPT by means of the hyperglycemic clamp technique. In addition, 15 sex- and age-matched control

Hyperparathyroidism: is it really the major factor affecting glucose tolerance in uremia?

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The effects of secondary hyperparathyroidism (sHPTH) on immunoreactive insulin (IRI) release and glucose (G) tolerance were studied in two groups of dialysis patients with normal (NPTH, n = 9) or elevated PTH levels (HPTH, n = 8), 27 +/- 24 and 660 +/- 440 pg/ml, respectively. The patients received

Carbohydrate metabolism in uremia.

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Abnormalities of insulin and glucose metabolism, namely glucose intolerance, inhibition of insulin secretion and insulin resistance, are present in children with chronic renal failure. Insulin resistance is universal among children with end-stage renal disease and may be caused by uremic toxins

Primary hyperparathyroidism, insulin resistance, and cardiovascular disease: a review.

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The presentation of primary hyperthyroidism (PHPT) has changed substantially in the last decade. Before the introduction of routine calcium measurement in most automated biochemistry serum analyzers, it usually was diagnosed after renal and bony lesions already were present. Nowadays, its

Lectin-binding sites in human parathyroid tissue.

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The aim of this study was to demonstrate several lectin-binding sites in human parathyroid tissue and to correlate these results with functional activity. The following lectins were tested for binding sites with certain carbohydrates (in parentheses): Arachis hypogea (PNA) (galactose), Ulex

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