intracranial hemorrhages/hypoxie

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Intracranial hemorrhages in kittens: hypernatremia versus hypoxia.

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Although CNS hemorrhages have long been observed in infants with hyaline membrane disease, the etiology of these hemorrhages is still unknown. Two proposed etiologies are hypoxia with acidosis and iatrogenic hypernatremia secondary to sodium bicarbonate therapy. An experiment on kittens comparing

Apneic oxygenation is associated with a reduction in the incidence of hypoxemia during the RSI of patients with intracranial hemorrhage in the emergency department.

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Critically ill patients undergoing emergent intubation are at risk of oxygen desaturation during the management of their airway. Patients with intracranial hemorrhage (ICH) are particularly susceptible to the detrimental effects of hypoxemia. Apneic oxygenation (AP OX) may be able to reduce the

Does hypoxia affect intensive care unit delirium or long-term cognitive impairment after multiple trauma without intracranial hemorrhage?

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BACKGROUND Within the traumatic brain injury population, outcomes are affected by hypoxic events in the early injury period. Previous work shows a high prevalence of cognitive deficits in patients with multiple injuries who do not have intracranial hemorrhage identified on admission head computed

Early hypoxemia burden is strongly associated with severe intracranial hemorrhage in preterm infants.

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OBJECTIVE The objective of this study was to define the association between the burden of severe hypoxemia (SpO2 ≤70%) in the first week of life and development of severe ICH (grade III/IV) in preterm infants. METHODS Infants born at <32 weeks or weighing <1500 g underwent prospective SpO2 recording

[Contribution to the electroencephalographic study of anoxia and intracranial hemorrhage in the newborn].

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[A particular type of anoxia of the newborn directly in relation with a massive intracranial hemorrhage].

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[Ultrasonography in the diagnosis of sequelae of perinatal anoxia and intracranial hemorrhage].

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[Influence of hypoxia on intracranial hemorrhage of perinatally expired newborn infants].

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[Causes and clinical manifestations of intracranial hemorrhages and hypoxia of the central nervous system in full-term newborn infants in fatal perinatal injuries].

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Maternal, fetal, and newborn complications associated with newborn intracranial hemorrhage.

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Two hundred twenty newborn infants with one or more fetal or newborn complications and 54 newborn infants without fetal or newborn complications were prospectively studied to assess the relationship between maternal, obstetric, fetal, and newborn complications and intracranial hemorrhage.

[The relationship between ligation of the ductus arteriosus and intracranial hemorrhage in preterm infants].

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Surgical closure of patent ductus arteriosus (PDA) and perioperative time have been proposed as conditions of increased risk of peri-intraventricular hemorrhage (PVH-IVH) in preterm infants. We examined by pre- and postoperative ultrasound (US) scan 15 low birth-weight neonates who underwent PDA

Brainstem auditory evoked potentials in very-low-birth-weight neonates with intracranial hemorrhage.

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A study was conducted in very-low-birth-weight (VLBW) neonates to correlate structural damage to the central nervous system due to intracranial hemorrhage (ICH) with electrophysiological function of the lower auditory system as measured by brainstem auditory evoked potentials (BAEP). BAEP testing of

Intracranial hemorrhage in the premature. Its predictive features and outcome.

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Sixty-four infants with birth weights of 500 to 1,500 g were studied to determine the incidence and outcome of intracranial hemorrhage. Thirty-seven (58%) had hemorrhage and of these 60% died. Of the survivors, progressive hydrocephalus requiring treatment developed in only two infants. Serial

Intracranial hemorrhage following administration of sodium bicarbonate in rabbits.

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Studies on the correlation between hyperosmolality and brain damage, especially intracranial hemorrhage, were carried out on young and newborn rabbits following infusion with 7% sodium bicarbonate. 1) All the young rabbits injected with 7% sodium bicarbonate died of hyperosmolality at over 380

Aminocaproic acid decreases the incidence of intracranial hemorrhage and other hemorrhagic complications of ECMO.

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Since the inception of extracorporeal membrane oxygenation (ECMO), hemorrhage has been a major complication often limiting its usefulness. This study was undertaken to evaluate the effect of aminocaproic acid (AMICAR), an inhibitor of fibrinolysis, on all hemorrhagic complications of ECMO including

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