leukocytosis/epileptische aanval

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Transient peripheral leukocytosis in children with afebrile seizures.

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The purpose of this study was (1) to demonstrate whether peripheral blood leukocytosis accompanies first afebrile seizures without bacterial infection, (2) to investigate the duration of leukocytosis, and (3) to assess the relationship between peripheral blood leukocytosis and seizure

Cerebrospinal Fluid Pleocytosis in Critical Care Patients With Seizures.

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OBJECTIVE To assess the etiology of cerebrospinal fluid (CSF) pleocytosis in critical care patients with seizure(s) or status epilepticus (SE). Many previous studies, some performed decades ago, concluded that CSF pleocytosis may be entirely attributable to seizure activity. METHODS We undertook a

Potential use of leukocytosis and anion gap elevation in differentiating psychogenic nonepileptic seizures from epileptic seizures.

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Epileptic seizures (ES) and psychogenic nonepileptic seizures (PNES) can be difficult to differentiate from each other in the emergency department (ED) setting. We have previously shown that the anion gap (AG) can help differentiate between ES and PNES in the ED. In this study, we explored whether

[A case of theophylline-associated seizures with postictal fever and postictal pleocytosis].

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We report a 70-year-old man who developed theophylline-associated seizure with postictal fever and postictal pleocytosis. He was admitted to our hospital for a left hemiconvulsion lasting for two hours. The concentration of theophylline was high (21.6ng/ml), and electroencephalogram demonstrated

Peripheral leukocytosis in children with febrile seizures.

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Peripheral blood leukocytosis has been reported following febrile seizures as a result of infection, the seizure, or both. To examine this relationship, 238 consecutive children < 5 years of age who experienced their first febrile seizure were evaluated. Lumbar punctures were electively done on 128

The duration of febrile seizures and peripheral leukocytosis.

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In 203 consecutive children with febrile seizures, no clear association (odds ratio = 1.0 [95% CI, 0.9-1.1], P = .59) was found between seizure duration and blood leukocytosis (> or = 15.0 x 10(9) cells/L). Increased leukocyte counts may be misinterpreted because of seizure duration. In children

Cerebrospinal fluid pleocytosis after seizures.

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Cerebrospinal fluid (CSF) pleocytosis after seizure activity has been anecdotally reported for many years, but it has not been well documented. We reviewed the records of all adult patients admitted to Grady Memorial Hospital from November 1979 through October 1980 with the diagnosis of seizure. Of

How often does a CSF pleocytosis follow generalized convulsions?

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Postictal pleocytosis is a transient and otherwise unexplained increase in cerebrospinal fluid (CSF) leukocytes occurring after repetitive generalized tonic-clonic seizures. We reviewed 98 episodes of seizures in 91 patients in whom the CSF was examined within 72 hours of the last seizure. Patients

Cerebrospinal fluid pleocytosis following simple, complex partial, and generalized tonic-clonic seizures.

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We observed postictal pleocytosis in 7 of 62 cerebrospinal fluid specimens obtained from 27 patients with epilepsy. Each patient had a known seizure disorder; none had any other cause for the pleocytosis. The maximum number of leukocytes was 12/mm3; the maximum number of erythrocytes was 190/mm3.

Febrile seizures with leukocytosis as a predictor for occult bacteremia.

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BACKGROUND

Febrile children 3-36 months old, who had a body temperature >39°C and white blood cell (WBC) count >15 000/mm³ were known to be at risk for occult pneumococcal bacteremia (OPB) in the pre-pneumococcal conjugate vaccine (PCV) era. The positive

[Transitory cerebrospinal fluid pleocytosis in a period of increased incidence of epileptic seizures (author's transl)].

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Question 1. Do febrile convulsions cause CSF pleocytosis?

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[Clinical and experimental studies on leukocytosis in cardiazol induced convulsions; studies on a central nervous system induced leukocytosis].

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[Spinal pleocytosis after generalized seizures].

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Piperacillin/tazobactam-induced seizure rapidly reversed by high flux hemodialysis in a patient on peritoneal dialysis.

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Despite popular use of piperacillin, the dire neurotoxicity associated with piperacillin still goes unrecognized, leading to a delay in appropriate management. We report a 57-year-old woman with end-stage renal disease receiving continuous ambulatory peritoneal dialysis (CAPD), who developed slurred

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