methemoglobinemia/braken

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Severe methemoglobinemia linked to gel-type topical benzocaine use: a case report.

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BACKGROUND Methemoglobinemia is an uncommon cause of tissue hypoxia, but it can be life threatening if it is not identified and treated promptly. OBJECTIVE To highlight the importance of understanding the potential risks of over-the-counter medications, especially in unsupervised use. Topical

Identical twin Hispanic male infants with nonbilious nonbloody vomiting and diarrhea.

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We present a case of twin Hispanic male infants fed with cow's milk formula who presented at 3 weeks of life with nonbilious, nonbloody vomiting and diarrhea. Laboratory evaluation revealed leukocytosis, acidosis, and methemoglobinemia. Sepsis evaluation was negative. Although they recovered quickly

Topical benzocaine (Hurricaine) induced methemoglobinemia during endoscopic procedures in gastric bypass patients.

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BACKGROUND Methemoglobinemia results from oxidation of ferrous to ferric iron in hemoglobin. In addition to a functional anemia, methhemoglobinemia causes the O2-binding affinity of the remaining O2 sites in the hemoglobin tetramer to increase; essentially shifting the oxyhemoglobin dissociation

Naphthalene Toxicity: Methemoglobinemia and Acute Intravascular Hemolysis.

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Naphthalene poisoning is a rare form of toxicity that may occur after ingestion, inhalation, or dermal exposure to naphthalene-containing compounds such as mothballs. Clinically, patients present with acute onset of dark brown urine, watery diarrhea, and non-bloody bilious vomiting 48-96 hours after

First case of methemoglobinemia caused by a ClO2-based household product.

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When new household products are developed and distributed, new injuries often occur in children. We report the first known case of methemoglobinemia caused by a chlorine dioxide (ClO2)-based household product. A 1-year-old boy presented to the emergency department with vomiting and poor complexion

[Acute dapsone exposure and methemoglobinemia in children: treatment with multiple doses of activated charcoal with or without the administration of methylene blue]

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OBJECTIVE: To study the changes in methemoglobinemia of 17 children admitted with acute exposure to dapsone complicated by a methemoglobin concentration greater than 20% of the total hemoglobin. The children were treated with multiple doses of activated charcoal with or without the administration of

Methemoglobinemia in an infant--Wisconsin, 1992.

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Methemoglobinemia among infants is a rare and potentially fatal condition caused by genetic enzyme deficiencies, metabolic acidosis, and exposure to certain drugs and chemicals. The most widely recognized environmental cause of this problem is ingestion of nitrate-containing water. Ingestion of

Methemoglobinemia associated with dermal application of benzocaine cream in a cat.

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Benzocaine, an ester-type local anesthetic, was believed responsible for apparent methemoglobinemia in a cat. The cat was admitted with acute respiratory distress, vomiting, and collapse, which began 15 to 20 minutes after topical application of the drug. Treatment consisted of supportive therapy

Methemoglobinemia following metoclopramide therapy in an infant.

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An infant with persistent vomiting was found to have methemoglobinemia. Review of his history revealed that he had been treated with metoclopramide. No other etiology of the methemoglobinemia was identified. It is concluded that the use of metoclopramide in a sick neonate can cause transient

Food Protein-Induced Enterocolitis Syndrome Causing Hypovolemic Shock and Methemoglobinemia.

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A 5-week-old previously healthy male presented with vomiting and diarrhea leading to hypovolemic shock and profound metabolic acidosis. He was subsequently found to have severe methemoglobinemia. The acidosis and shock improved with fluid resuscitation and methemoglobinemia was successfully treated

Incidence of subclinical methemoglobinemia in infants with diarrhea.

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OBJECTIVE Infants with diarrhea are at a greater-than-recognized risk of developing methemoglobinemia. METHODS Prospective clinical study. METHODS A university hospital pediatric emergency department. METHODS Consecutive infants under 6 months of age with a history of diarrhea of more than 24 hours'

Boilerbaisse: an outbreak of methemoglobinemia in New Jersey in 1992.

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BACKGROUND On October 20, 1992, > 40 children from one elementary school visited the school nurse due to the acute onset of blue lips and hands, vomiting, and headache during and after the school lunch periods. Forty-nine children were seen by physicians that day and 14 were hospitalized. Laboratory

Prolonged hemolysis and methemoglobinemia following organic copper fungicide ingestion.

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Acute ingestion of copper sulfate has been reported to cause gastrointestinal injury, hemolysis, methemoglobinemia, hepatorenal failure, shock; or even death. The toxicity of organocopper compounds, however, remains largely unknown. A 40-y-old man attempted suicide by ingesting some 50 ml of

[Food protein-induced enterocolitis syndrome: A case report of diarrhea with hypovolemic shock and methemoglobinemia].

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We report on the case of a young infant with chronic diarrhea that worsened and turned into hypovolemic shock with methemoglobinemia. We underline and discuss the main features of food protein-induced enterocolitis syndrome (FPIES). FPIES is a non-IgE-mediated food allergy involving tumor necrosis

Acquired Methemoglobinemia - A Sporadic Holi Disaster.

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OBJECTIVE To study clinical profile and outcome in patients with methemoglobinemia following exposure to toxic colors during Holi festival. METHODS This retrospective study included 112 children (5 to 12 years) admitted with methemoglobinemia after playing Holi. Clinical and treatment details were

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