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nephrosis/carbohydrate
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Carbohydrate metabolism in expertmental nephrosis.
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Structural changes in the protein and carbohydrate components of glomerular basement membrane in aminonucleoside nephrosis.
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[Variations in carbohydrate metabolism in alloxan diabetes in the rabbit and glycogen nephrosis of the renal tubular epithelium].
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[Paper electrophoresis of clinical development of lipoid nephrosis, proteins, carbohydrates and lipids].
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Metabolic channeling in experimental nephrosis. I. Protein and carbohydrate metabolism.
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Treatment of experimental nephrosis by culture filtrate of Cryptococcus neoformans var. gattii (CneF).
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The therapeutic effect of the culture filtrate of cryptococcus neoformans var. gattii (CneF) was tested in Adriamycin-induced nephropathy. The CneF was administered at different doses (36, 54 and 90 mg/kg based on carbohydrate concentration), one i.p. injection every 72 hours for a total of 10
A new leukocyte hyperadhesion syndrome of delayed cord separation, skin infection, and nephrosis.
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Leukocyte adhesion deficiency (LAD) I is a well-described genetic disorder in which leukocytes are unable to migrate to sites of inflammation due to mutations in the ITGB2 gene coding for the β subunit of β2 (CD18) leukocyte integrins. The classic symptoms of the disease present in the newborn
Clinical improvement of membranous nephropathy after endoscopic resection of double early gastrointestinal cancers.
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A-52-year-old man presented to our hospital with nephrotic syndrome caused by membranous nephropathy. Early gastric adenocarcinoma confined to the submucosa, and well-differentiated adenocarcinoma in a sigmoid adenoma were detected by screening endoscopy. Two years after complete endoscopic
Clinical and biochemical signs in Fleckvieh cattle with genetically confirmed Fanconi-Bickel syndrome (cattle homozygous for Fleckvieh haplotype 2).
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Fanconi-Bickel Syndrome (FBS) is an autosomal recessive disorder of the carbohydrate metabolism, which has been reported in human and some animals (OMIA 000366-9913). In Fleckvieh cattle it is caused by mutations in SLC2A2, a gene encoding for glucose transporter protein 2 (GLUT2), which is
Effects of a dietary load of acid or base on changes induced by lactose in rats.
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Feeding lactose or other slowly digestible carbohydrates to adult mammals may induce a variety of effects including hyperplasia and neoplasia. The most fundamental effect probably is the increased production in the large intestine of short-chain fatty acids (SCFA) resulting from increased
INFLUENCE OF DIETARY FACTORS AND SEX ON THE TOXICITY OF CARBON TETRACHLORIDE IN RATS.
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Six groups of rats on different diets were exposed to the inhalation of carbon tetrachloride (about 300 p. p. m.) for 150 days. Food intake and changes in weight were followed throughout the experiment. Animals fed a diet low in protein showed greater susceptibility than rats on a diet high in
[Idiopathic nephrotic syndrome].
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Nephrotic syndrome (NS) is defined by massive proteinuria and hypoalbuminemia, with resulting hyperlipidemia and edema. The most common cause of NS in children is idiopathic nephrotic syndrome (INS), also called nephrosis. Its annual incidence has been estimated to 1-4 per 100,000 children and
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