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phlebitis/glutathione
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Antioxidant supplementation improves platelet membrane fluidity in idiopathic retinal periphlebitis (Eales' disease).
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OBJECTIVE
Oxidative damage to cellular membranes plays an important role in the pathobiology of tissue injury. Retinal photoreceptors and platelets are an easy target of oxidants because of high proportion of polyunsaturated fatty acids. A tertiary-care center-based prospective study was undertaken
[Pharmacological study and pharmaceutical intervention to reduce intravenous injection-induced vascular injury].
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Intravenous injection often causes vascular injury such as venous irritation, vascular pain, and phlebitis. Vascular injury deteriorates the patient's QOL and sometimes limits the continuation of injection therapy. Pharmaceutical intervention and pharmacological mechanisms used to reduce vascular
Eales' disease: oxidant stress and weak antioxidant defence.
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Eales' disease (ED) is an idiopathic retinal periphlebitis characterized by capillary non-perfusion and neovascularization. In addition to the existing system, a new staging system has been proposed by Saxena et al. Immunological, molecular biological and biochemical studies have indicated the role
Activation of p38 MAPK by oxidative stress underlying epirubicin-induced vascular endothelial cell injury.
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Epirubicin, an anthracycline antitumor drug, often causes vascular injury such as vascular pain, phlebitis, and necrotizing vasculitis. However, an effective prevention for the epirubicin-induced vascular injury has not been established. The purpose of this study is to identify the mechanisms of
Role of oxidative stress in vinorelbine-induced vascular endothelial cell injury.
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Vinorelbine (VNR), a vinca alkaloid anticancer drug, often causes vascular injury such as venous irritation, vascular pain, phlebitis, and necrotizing vasculitis. The purpose of this study was to identify the mechanisms that mediate the cell injury induced by VNR in porcine aorta endothelial cells
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