potassium chloride/necrotisch

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Tumor necrosis factor-alpha triggers cell death of sensitized potassium chloride-stimulated cholinergic neurons.

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Cell death of cholinergic neurons of the basal forebrain plays an important role in neurodegenerative disorders, such as Alzheimer's disease. Inflammatory cytokines, such as, for example, tumor necrosis factor-alpha (TNF-alpha), may be involved in these neurodegenerative processes. The aim of this

[Tissue necrosis after accidental perivenous injection of potassium chloride].

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Accidental subcutaneous infiltration of potassium chloride solution causing necrosis.

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[Effect of potassium chloride and magnesium chloride on electrocardiographic and morphological changes in ischemic necrosis of the myocardium].

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[Skin necrosis resulting from the extravasation of intravenous injections].

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Extravasation is the diffusion of a product injected intravenously into the perivascular or subcutaneous spaces. Skin necrosis of iatrogenic origin can be observed for example with a subcutaneous perfusion of solution containing potassium chloride or the extravasation of 30% hypertonic glucose

Lidocaine treatment of experimental cutaneous lesions from potassium chloride injection.

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Potassium chloride (KCl) given subcutaneously in high concentrations causes necrosis of skin, possibly from vasoconstriction around the injection site. The authors studied guinea pigs given subcutaneous injections of various volumes and concentrations of KCl and observed the severity of the

Paraplegia following epidural potassium chloride.

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A patient is presented in whom 15 ml of 15% potassium chloride (30 mM) mixed with bupivacaine was injected epidurally for relief of pain resulting from widespread pelvic malignancy. Within minutes the patient complained of severe pain in both the lower limbs and rapidly became paraplegic with

Medication-induced acute esophageal necrosis: a case report.

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BACKGROUND Acute esophageal necrosis or Gurvits syndrome is a rare clinical condition characterized by necrotic esophageal mucosa with an abrupt end at the gastroesophageal junction. Its etiology is multifactorial, but mainly related to low-flow states. We describe a case in which a patient

Metastatic breast carcinoma induces vascular endothelial dysfunction in Balb-c mice: Role of the tumor necrosis factor-α and NADPH oxidase.

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Although the oxidative stress and inflammation are closely related with breast cancer, there is no study directly examining the possible changes in vascular functions in the presence of breast carcinoma. The goal of the present study was to evaluate changes in vascular reactivity in tumor-bearing

Clinical features and management of poisoning due to potassium chloride.

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Potassium is one of the most abundant ions in the human body and yet it is difficult to assess potassium balance. Potassium chloride is extensively used as a potassium supplement, both by physicians as a therapeutic modality and by the general public, mostly in the form of salt substitute.

Resveratrol Normalizes the Deterioration of Smooth Muscle Contractility after Intestinal Ischemia and Reperfusion in Rats Associated With an Antioxidative Effect and Modulating Tumor Necrosis Factor Alpha Activity.

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The aim of the present study was to investigate the effect of resveratrol on cytokine levels and oxidative stress in intestinal ischemia/reperfusion (I/R) injuries.To induce intestinal I/R, the superior mesenteric artery was occluded for 30 min and then

Acute toxicity of sodium nitrate, potassium nitrate, and potassium chloride and their effects on the hemolymph composition and gill structure of early juvenile blue swimmer crabs(Portunus pelagicus Linnaeus, 1758) (Decapoda, Brachyura, Portunidae).

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Various nutrients, including K+ and NO3-, are increasingly being discharged into aquatic systems via anthropogenic sources, which may impact marine organisms. The present study was conducted on blue swimmer crab (Portunus pelagicus) early juveniles to determine the acute toxicity of NaNO3, KNO3, and

Novel Modification of Potassium Chloride Induced Cardiac Arrest Model for Aged Mice.

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Experimental cardiac arrest (CA) in aging research is infrequently studied in part due to the limitation of animal models. We aimed to develop an easily performed mouse CA model to meet this need. A standard mouse KCl-induced CA model using chest compressions and intravenous epinephrine for

Acute kidney injury in human leptospirosis: an immunohistochemical study with pathophysiological correlation.

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Tubulointerstitial nephritis is a common clinicopathological finding in leptospirosis. Clinically, nonoliguric acute kidney injury (AKI), hypokalemia, sodium, and magnesium wasting frequently occur in leptospirosis. The exact mechanisms of renal involvement remain largely unclear.

Ephedra-Induced Gastric Mucosal Injury.

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In this case report we detail the difficult case of a 74-year-old male patient who was transferred to our cardiac intensive care unit in severe shock with an acute abdomen. His abdomen was emergently explored revealing isolated gastric necrosis and ischemia. A subtotal gastrectomy was performed and

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