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pulmonary fibrosis/nicotine
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Bladzijde 1 van 108 resultaten
Tobacco smoke-induced lung fibrosis and emphysema.
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Despite public health campaigns discouraging smoking, 1,000 American children every day become smokers, ensuring that tobacco-related health complications will be with us for decades to come. Smoking is the greatest risk factor for both chronic obstructive lung disease and interstitial lung disease.
Maternal nicotine exposure exacerbates neonatal hyperoxia-induced lung fibrosis in rats.
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BACKGROUND
Maternal nicotine exposure increases lung collagen in fetal and newborn animals. Connective tissue growth factor (CTGF) plays a role in hyperoxia-induced pulmonary fibrosis.
OBJECTIVE
To determine whether pre- and postnatal nicotine exposure can augment CTGF expression and postnatal
CB2R agonist prevents nicotine induced lung fibrosis.
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Developmental nicotine exposure results in programming of alveolar simplification and interstitial pulmonary fibrosis in adult male rats.
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OBJECTIVE
To determine the long-term effect of maternal nicotine intake on the lung development of the offspring in adult life, we analyzed the alveolar structure, protein expression in the adult rat offspring lungs.
METHODS
We determined animal body weight (BW), lung weight (LW), lung/body weight
Cardiac-specific overexpression of metallothionein rescues nicotine-induced cardiac contractile dysfunction and interstitial fibrosis.
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Cigarette smoking is a devastating risk factor for cardiovascular diseases and nicotine is believed the main toxin component responsible for the toxic myocardial effects of smoking. Nonetheless, neither the precise mechanism of nicotine-induced cardiac dysfunction nor effective treatment is
Diffuse pulmonary fibrosis and blackfat-tobacco smoking in Guyana.
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[Pulmonary fibrosis caused by tobacco].
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[Combination of pulmonary fibrosis and emphysema: Is tobacco once again the protagonist?].
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[Diffuse interstitial pulmonary fibrosis in tobacco-industry workers].
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Efficacy and Safety of Nintedanib in Idiopathic Pulmonary Fibrosis, Cytisine versus Nicotine for Smoking Cessation, and FACED Score for Non-Cystic Fibrosis Bronchiectasis.
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Serum angiotensin-converting enzyme and lysosomal enzymes in tobacco workers.
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The activities of angiotensin-converting enzyme (ACE) in people with extrinsic allergic alveolitis (EAA) have been both increased and decreased. These observations suggest that pulmonary macrophages or endothelial cells participate in the disease process. Exposure to molds in the tobacco industry
Distribution of emphysema and fibrosis in idiopathic pulmonary fibrosis with coexisting emphysema.
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Lung bullae and pulmonary fibrosis associated with marijuana smoking.
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A 26-year-old man with a history of heavy marijuana and minimal tobacco use was found to have extensive bilateral lung bullae and interstitial fibrosis, heavily infiltrated by pigmented macrophages. These features can be associated with marijuana smoking. The differential diagnoses in this patient
Chronic nicotine in hearts with healed ventricular myocardial infarction promotes atrial flutter that resembles typical human atrial flutter.
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The potential of chronic nicotine exposure for atrial fibrillation (AF) and atrial flutter (AFL) in hearts with and without chronic myocardial infarction (MI) remains poorly explored. MI was created in dogs by permanent occlusion of the left anterior descending coronary artery, and dogs were
Characteristics of tobacco-related lung diseases in Fukuoka Prefecture, Japan: A prospective, multi-institutional, observational study.
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