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Bladzijde 1 van 315 resultaten
Right insular atrophy in neurocardiogenic syncope: a volumetric MRI study.
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OBJECTIVE
Alterations in the central autonomic network are hypothesized to play a role in the pathophysiologic mechanism underlying neurocardiogenic syncope; however, few data are available regarding the structural changes of the brain in this condition. We used voxel-based morphometry and regional
Enzootic fatal syncope (toxic heart degeneration) in fowls.
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Drop attacks and instability of the degenerate cervical spine.
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[Progressive gait instability and weakness with recurrence presyncopes/syncopes. Multiple system atrophy].
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[Statistical survey of the changes in atrioventricular conduction after injection of ajmaline base in the case of 45 patients unaffected by syncopes or serious deterioration in conduction (author's transl)].
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[Fainting at rising, locomotion disorder, aphasia and an increase in muscle tonus (autonomic nervous function)--stomach cancer: (Shy-Drager syndrome) (multiple system atrophy)].
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Autonomic predominant multiple system atrophy in the context of Parkinsonian and cerebellar variants.
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OBJECTIVE
Autonomic dysfunction is often a late feature of multiple system atrophy (MSA). Based on early features, MSAp (Parkinsonian) and MSAc (cerebellar) variants are known. We studied the severity of autonomic dysfunction at first examination in MSA patient that may have relevance in its early
[An autopsy case of multiple system atrophy presenting with rapid progression of autonomic disturbance].
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We report an autopsy case of multiple system atrophy (MSA) presenting with rapid progression of autonomic disturbance. He was admitted to our hospital because of gait disturbance and dysarthria. The patient was a Japanese man, who first noticed gait disturbance and dysarthria at age 58, followed by
Automated external defibrillator documented degeneration of pre-excited atrial fibrillation into ventricular fibrillation.
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Wolff-Parkinson-White (WPW) syndrome can be the cause of syncope or sudden cardiac death, which results from ventricular fibrillation (VF) degenerated from rapid anterograde conduction of atrial fibrillation (AF) to the ventricles through the accessory pathway. We present a case of WPW syndrome in
Prinzmetal angina in the differential diagnosis of syncope.
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Prinzmetal (variant) angina may be associated with cardiac arrhythmias that can deteriorate to fatal ventricular arrhythmias. We present 2 patients with syncope where vasospastic angina and severe ventricular arrhythmias were found to be responsible for the syncopal episodes.
Usefulness of tilt-induced heart rate changes in the differential diagnosis of vasovagal syncope and chronic autonomic failure.
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OBJECTIVE
To determine whether the heart rate changes during tilt table testing could be used in the differential diagnosis between vasovagal syncope and chronic autonomic failure.
METHODS
We compared the relationship between electrocardiographic R-R intervals and beat-to-beat blood pressure in 43
Ivabradine in treatment of sinus tachycardia mediated vasovagal syncope.
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OBJECTIVE
Ivabradine, an I(f) current blocker, has shown promising results in treatment of postural orthostatic tachycardia syndrome (POTS). There is a subgroup of vasovagal syncope (VVS) patients, who demonstrate sinus tachycardia before collapse on tilt testing mimicking some features of POTS.
A retrospective study of 153 cases of undiagnosed collapse, syncope or exercise intolerance: the outcomes.
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OBJECTIVE
To retrospectively assess the long-term outcome for dogs that were presented with collapse, syncope or exercise intolerance for which an underlying cause is not identified.
METHODS
The clinical records of animals presenting to two practices were assessed. Dogs which presented for collapse,
Pacing rate increase as cause of syncope in a patient with severe cardiomyopathy.
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Hemodynamic deterioration occurs with ventricular pacing rate increase in the presence of severe chagasic cardiomyopathy. Syncope and orthopnea occurred during ventricular pacemaker evaluation when the pacing rate was temporarily increased by magnet application. Cardiac output decreased by 54%, the
Otolithic membrane damage in patients with endolymphatic hydrops and drop attacks.
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OBJECTIVE
1. Evaluate the otolithic membrane in patients with endolymphatic hydrops (EH) and vestibular drop attacks (VDA) undergoing ablative labyrinthectomy. 2. Correlate intraoperative findings to archival temporal bone specimens of patients with EH.
METHODS
Retrospective case
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