uremia/hypoxie

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Uremia and Hypoxia Independently Induce Eryptosis and Erythrocyte Redox Imbalance.

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Red blood cell (RBC) death could contribute to anemia in chronic kidney disease (CKD) patients. Recent observational research has suggested a relationship between RBC death (eryptosis) and hypoxemia in hemodialysis patients. Thus, we studied the isolated and joint effects of a uremic

Uremia induces abnormal oxygen consumption in tubules and aggravates chronic hypoxia of the kidney via oxidative stress.

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In addition to causing uremic symptoms, uremic toxins accelerate the progression of renal failure. To elucidate the pathophysiology of uremic states, we investigated the effect of indoxyl sulfate (IS), a representative uremic toxin, on oxygen metabolism in tubular cells. We demonstrated an increase

Indoxyl sulfate signals for rapid mRNA stabilization of Cbp/p300-interacting transactivator with Glu/Asp-rich carboxy-terminal domain 2 (CITED2) and suppresses the expression of hypoxia-inducible genes in experimental CKD and uremia.

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Chronic hypoxia in the tubulointerstitium serves as a final common pathway in progressive renal disease. Circumstantial evidence suggests that hypoxia-inducible factor (HIF)-1 in the ischemic tubules may be functionally inhibited in a chronic kidney disease (CKD) milieu. In this study, we

Refractoriness of the right atrium and A--V conducting system. Influence of practolol, chronic hypoxia and uremia.

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[Centralization of the circulation, hypoxia and uremia following pulmonary embolism].

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Renal anoxia and the traumatic uraemia syndrome.

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Effect of uremia and its treatment on pulmonary function.

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Alterations in respiratory drive, mechanics, muscle function, and gas exchange are frequent if not invariable consequences of uremia. Pulmonary dysfunction may be the direct result of circulating uremic toxins or may result indirectly from volume overload, anemia, immune suppression, extraosseous

Hypoxia: A cause of acute renal failure and alteration of gastrointestinal microbial ecology.

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Oxygen is very important to the existence of life. Oxygen deficiency, defined as hypoxia, elicits adaptive responses in cells and tissues. Lower oxygen concentration can cause the alteration of renal function, affects the maintenance of a balance of the body fluids, electrolytes, pH, and blood

Uremia induces functional incompetence of bone marrow-derived stromal cells.

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BACKGROUND Chronic kidney disease (CKD) is associated with increased risk for cardiovascular diseases (CVD). We hypothesized that inadequate angiogenic response in uremic patients could result from dysfunction of bone marrow-derived stromal cells [mesenchymal stem cells (MSCs)]. METHODS We

Acid-base status and blood gas alterations following experimental uremia in cattle.

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Experimental uremia created by obstructing the urethra of 7 cross-bred bulls was associated with significant (P less than 0.05) increases in PCV, blood urea nitrogen concentration, arterial and venous pH and PCO2, arterial bicarbonate, and base excess. Total serum protein concentration decreased

Clinical characteristics of patients with uremia undergoing maintenance hemodialysis complicated with COVID-19

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This study aimed to evaluate the onset characteristics of patients with uremia undergoing maintenance hemodialysis complicated with COVID-19, so as to improve the understanding, diagnosis, and treatment.26 cases were confirmed cases of COVID-19. Confirmed patients with COVID-19 undergoing

[Effect of hemodialysis on the functional status of small airways and partial pressure of oxygen and carbon dioxide in the blood of patients with chronic uremia].

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In 18 patients with chronic uremia the effect of 4 hour hemodialysis on VC, FEV1, PEF, MEF25, MEF50, MEF75, PaO2 and PaCO2 were analysed. Significant falls in PaO2 and PaCO2 were found after ending the four hour long hemodialysis. Only nonsignificant changes in the other parameters were observed.

The effect of hemodialysis on lung function, gas exchange, and response to carbon dioxide stimulation in chronic uremia.

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Little is known of the effect of chronic hemodialysis on lung function. In six patients with chronic uremia, we performed pulmonary function tests, sampled arterial blood, and measured the ventilatory response to carbon dioxide inhalation before and after routine hemodialysis. Patients were in the

[Leukopenia and hypoxemia induced by hemodialysis].

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Patients with uremia usually develop hypoxemia during hemodialysis therapy. It has been thought by most of the investigators that the development of hypoxemia is related to the impropriety of biocompatibility of the dialyzer membranes. Besides, acetate dialysate through metabolism may cause a

Impact of nocturnal hemodialysis on the variability of heart rate and duration of hypoxemia during sleep.

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BACKGROUND Nocturnal hemodialysis (NHD) alleviates uremia-related sleep apnea, a condition characterized by increased sympathetic activity and diminished heart rate (HR) variability. We tested the hypothesis that NHD reduces both hypoxemia and sympathetic neural contributions to HR variability

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