lactate dehydrogenase/seizures

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[Lactate dehydrogenase isoenzymatic activity in different parts of the brain and in the blood serum of rats from different lines during a convulsive seizure caused by the action of a sound stimulus].

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Total lactate dehydrogenase activity and relative activities of LDH-1 and LDH-2 isozymes were found to be distinctly elevated in the cortex of cerebral hemispheres (locomotory region) and in medulla oblongata of audiogenic rats KM strain. The increase in the total LDH activity was also observed in

[Effect of audiogenic convulsions on the activity of n- and m-forms of lactate dehydrogenase in the neurons and neuroglia of different sections of central nervous system].

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It was shown spectrophotometrically that in Krushinsky-Molodkina and Wistar rats the ratio of the activity of the aerobic H-forms of lactic dehydrogenase (LDH) to the activity of the anaerobic M-forms was higher in the neurons of the cerebral cortex and the Purkinje's cells of the cerebellum and in

Cytospectrometric studies on the lactate dehydrogenase isoenzymes in functionally different neuron-neuroglia units. II. Species and strain differences and effects of histotoxic hypoxia and audiogenic convulsions.

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Cytospectrophotometric determination of H- and M-forms of LDH has shown that in white mice as compared with Wistar rats the activity ratio of H- to M-forms is lower in cerebral cortex neurons but higher in spinal cord motoneurons. No statistically significant species difference has been found in

Fermenting Seizures With Lactate Dehydrogenase.

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[Activity of aldolase, lactate dehydrogenase and content of lactic and pyroracemic acids in different areas of the brain in rat, with repeated convulsions evoked by corazole].

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[Observations on succinate and lactate dehydrogenase activity following cardiazol convulsions. (A histochemical study with microdensitometrical evaluation)].

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Citrate synthase and lactate dehydrogenase activities in rat cerebral cortex following the administration of the convulsants bicuculline and 3-mercaptopropionic acid.

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Systemic administration of bicuculline (Bic) and 3-mercaptopropionic acid (MP) induces generalized seizure in laboratory animals. The effect of these convulsants on lactate dehydrogenase and citrate synthase activities in rat cerebral cortex was studied. Bic was administered i.p. at 1.0 mg/kg

Protective effects of TRH and its stable analogue, RGH-2202, on kainate-induced seizures and neurotoxicity in rodents.

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Thyrotropin-releasing hormone (TRH) has been postulated to be involved in the regulation of seizures and neural degeneration. We examined the effects of TRH and its stable analogue, RGH-2202, on the kainate-induced seizures and excitotoxicity in mice - a model of a drug-resistant temporal lobe

Deletion of the Na/HCO3 Transporter NBCn1 Protects Hippocampal Neurons from NMDA-induced Seizures and Neurotoxicity in Mice.

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The Na/HCO₃ cotransporter NBCn1/SLC4A7 can affect glutamate neurotoxicity in primary cultures of rat hippocampal neurons. Here, we examined NMDA-induced neurotoxicity in NBCn1 knockout mice to determine whether a similar effect also occurs in the mouse brain. In primary cultures of

Diagnostic significance and source of lactate dehydrogenase and its isoenzymes in cerebrospinal fluid of children with a variety of neurological disorders.

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Two hundred and thirty-four cerebrospinal fluid (CSF) specimens from 183 different children were analysed for total lactate dehydrogenase (LD) activity and LD isoenzyme distribution. The LD activities were elevated in the CSF of patients with meningitis, especially with bacterial infections, and

[Cytophotometric determination of the relationship between the chief isoenzyme forms of lactate dehydrogenase in the neuron--neuroglia system in different fuctional states of the nervous system].

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In white mice, unlike the Wistar rats, the ratio of activities of H- and M-forms of lactate dehydrogenase (LDH) is lower in the cerebral cortex neurons but higher in the spinal cord motoneurons within the limits of sensitivity and correctness of the cytospectro-photometric method. No statistically

Altered activities of rat brain metabolic enzymes caused by pentylenetetrazol kindling and pentylenetetrazol--induced seizures.

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The aim of our study was to investigate amino acid and energy metabolism of pentylenetetrazol (PTZ)-kindled animals. Glutamate dehydrogenase, aspartate-aminotransferase (AST), alanine-aminotransferase, gamma-glutamyltransferase, alkaline phosphatase (ALP), lactate dehydrogenase (LDH) and creatine

Cerebrospinal fluid lactate dehydrogenase in infants with perinatal asphyxia.

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Cerebrospinal fluid (CSF) lactate dehydrogenase was determined in 19 control infants without asphyxia (Group I), 24 infants with perinatal asphyxia (Group II), and 26 asphyxiated infants with seizures (Group III). Mean birthweights, gestational ages, CSF glucose, protein and red blood cells, and the

In vivo alterations in lactate dehydrogenase (LDH) and LDH isoenzymes patterns by acute carbofuran intoxication.

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Intoxication with an acute dose of carbofuran (1.5 mg/kg, sc) in male Sprague-Dawley rats evoked severe toxic manifestations characteristic of hypercholinergic preponderance with profound muscle fasciculations and convulsions during 30-60 min, lasting for about 2 h. Lactate dehydrogenase, a

Spinal Fluid Lactate Dehydrogenase Level Differentiates between Structural and Metabolic Etiologies of Altered Mental Status in Children.

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OBJECTIVE Altered mental status is a common cause of intensive care unit admission in children. Differentiating structural causes of altered mental status from metabolic etiologies is of utmost importance in diagnostic approach and management of the patients. Among many biomarkers proposed to help

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