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Página 1 a partir de 90 resultados
Muscle adenine nucleotide degradation during submaximal treadmill exercise to fatigue.
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The aim was to investigate metabolic response in muscle during submaximal treadmill exercise to fatigue, with a special emphasis on adenine nucleotide degradation products such as inosine monophosphate (IMP) in muscle and hypoxanthine, xanthine and uric acid in plasma. Five Standardbred trotters
[Nicotinamide adenine dinucleotide (NADH) in patients with chronic fatigue syndrome].
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BACKGROUND
Nicotinamide adenine dinucleotide (NADH) may be depleted in chronic fatigue syndrome (SFC). The purpose of the study was to evaluate the efficacy of supplementation with NADH in these patients.
METHODS
A double blind, placebo controlled, 3 month long clinical trial was conducted. The
Comparison of oral nicotinamide adenine dinucleotide (NADH) versus conventional therapy for chronic fatigue syndrome.
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OBJECTIVE
To compare effectiveness of oral therapy with reduced nicotinamide adenine dinucleotide (NADH) to conventional modalities of treatment in patients with chronic fatigue syndrome (CFS).
BACKGROUND
CFS is a potentially disabling condition of unknown etiology. Although its clinical
Effect of coenzyme Q10 plus nicotinamide adenine dinucleotide supplementation on maximum heart rate after exercise testing in chronic fatigue syndrome - A randomized, controlled, double-blind trial.
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Chronic Fatigue Syndrome (CFS) is a complex condition, characterized by severe disabling fatigue with no known cause, no established diagnostic tests, and no universally effective treatment. Several studies have proposed symptomatic treatment with coenzyme Q10 (CoQ10) and nicotinamide adenine
Changes in adenine nucleotide, SOD and MDA contents and mitochondria ulteractructure during diaphragm fatigue in dogs induced by diaphragm pacing.
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OBJECTIVE: To explore the possible mechanisms for the occurrence of diaphragm fatigue induced by diaphragm pacing. METHODS: The phrenic nerve of 8 dogs were exposed and subjected to stimulation with electric pulses emitted by a diaphragm pacemaker to induce diaphragm fatigue models, and the contents
Chronic fatigue syndrome.
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BACKGROUND
Chronic fatigue syndrome (CFS) affects between 0.006% and 3% of the population depending on the criteria of definition used, with women being at higher risk than men.
METHODS
We conducted a systematic review and aimed to answer the following clinical question: What are the effects of
Chronic fatigue syndrome.
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BACKGROUND
Chronic fatigue syndrome (CFS) affects between 0.006% and 3% of the population depending on the criteria of definition used, with women being at higher risk than men.
METHODS
We conducted a systematic review and aimed to answer the following clinical question: What are the effects of
Artificial Oxygen Carrier Improves Fatigue-Resistance in Slow-muscle but not in Fast-muscle in a Rat in situ Model.
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Effects of liposome-encapsulated hemoglobin with high O2 -affinity (h-LEH), an artificial O2 carrier on skeletal muscle, were studied by in situ fatigue-resistance test in fast-type Plantaris (PLT) and slow-type Soleus (SOL) muscles with or without ischemia in the rat. The
Incremental treadmill exercise until onset of fatigue and its relationship to metabolic response and locomotion pattern.
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The aim was to study metabolic response and locomotion pattern in Standardbred trotters during incremental treadmill exercise performed by increasing speed by 1 m/s in 1 min steps (start 7 m/s) until the onset of fatigue. The test protocol included determination of oxygen uptake, heart rate (HR),
Frequency-dependent fatigue development during electrical stimulation in the masseter muscle of pigtail monkeys.
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Low-frequency fatigue was investigated in nine female and one male adult pigtail monkeys (Macaca nemestrina) with a mean weight of 5.3 kg (range 4.3 to 6.5 kg). After sedation and anesthesia, silver electrodes were inserted into the anterior and posterior parts of the right masseter muscle. The
Adenine nucleotide depletion in human muscle during exercise: causality and significance of AMP deamination.
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The predominant route for adenine nucleotide catabolism in skeletal muscle is deamination of AMP to inosine monophosphate (IMP) and ammonia (NH3). Deamination of AMP is enhanced during exercise when the capacity to rephosphorylate ADP is impaired. Thus, in human muscle the formation of IMP (NH3)
Effects of nicotinamide adenine dinucleotide hydride on physical and mental performance.
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The aim of this study was to assess the effects of nicotinamide adenine dinucleotide hydride (NADH) on maximal oxygen uptake (VO2max), maximal anaerobic running time, and mental performance. Eight men were exposed to a supplement treatment (30 mg NADH as ENACHI tablets per day) and to a placebo
Variation in the catalytic potential of AMP deaminase during muscular fatigue.
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Substrate- and co-factor-dependent kinetics of AMP deaminase were studied in normal and fatigued gastrocnemius muscles of frog. Normal muscle enzyme showed greater enzyme co-factor affinity than enzyme-substrate affinity as evinced by low Kp values. Fatigue phenomenon was found to decrease the
Impaired oxidative metabolism increases adenine nucleotide breakdown in McArdle's disease.
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Two patients with muscle phosphorylase deficiency [McArdle's disease (McA)] were studied during bicycle exercise at 40 (n = 2) and 60 W (n = 1). Peak heart rate was 170 and 162 beats/min, corresponding to approximately 90% of estimated maximal heart rate. Muscle samples were taken at rest and
The importance of ATPase microenvironment in muscle fatigue: a hypothesis.
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A broadly held opinion is that fatigue is not due to an insufficient supply of ATP to the energy consuming mechanisms because tissue [ATP] always remains at least one order of magnitude higher than Km for ATP of any ATPase. In general these findings also suggest that ATP consumption is well balanced
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