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aldolase/acidente vascular cerebral
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6 resultados
Aldolase C/zebrin II is released to the extracellular space after stroke and inhibits the network activity of cortical neurons.
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Cell death after stroke involves apoptotic, autophagocytic and necrotic mechanisms which may cause the release of cytosolic proteins to the extracellular space. Aldolase C (AldC) is the brain specific isoform of the glycolytic enzyme fructose-1,6-bisphosphate aldolase. According to its
Activity of creatine kinase, aminotransferases and aldolase in cerebral strokes.
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[Behavior of aldolase and lactic dehydrogenase activity of the cerebrospinal fluid in cerebral apoplexy].
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[The effect of magnesium sulfate used for killing rabbits on aldolase activity in organs].
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The effect of the system of mating and the way of killing on the aldolase activity in the tissue of rabbits was the task of this research work. The research work was realized in 201 New Zealand white rabbits aged 140 days, mated outbred and inbred. Two methods of killing were used: classical method,
Intranasal wnt-3a alleviates neuronal apoptosis in early brain injury post subarachnoid hemorrhage via the regulation of wnt target PPAN mediated by the moonlighting role of aldolase C.
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Neuronal apoptosis is one of the main pathophysiological events in the early brain injury (EBI) post subarachnoid hemorrhage (SAH). Wnt-3a, one of the endogenous wnt ligands crucial in neurogenesis, has been proven to be efficacious in neuroprotection in traumatic brain injury and ischemic stroke.
[Investigation of etiologies for acute renal failure due to rhabdomyolysis in 5 patients].
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We experienced 5 cases of acute renal failure due to rhabdomyolysis during the last two years and investigated those etiologies. Diagnosis of rhabdomyolysis was established by the detection of elevated serum creatine phosphokinase, myoglobin, aldolase, myoglobinuria as well as by the clinical
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