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amaurosis fugax/cefaleia
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A case report of isolated orbital vasculitis mimicking retinal migraine: A potential cause of recurrent transient monocular blindness and ipsilateral headache.
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Background Retinal migraine is an important differential diagnosis of recurrent transient monocular blindness accompanied by headache when other etiologies are excluded. Here, we report a case of orbital vasculitis which initially mimicked retinal migraine. Case report A 47-year-old woman had
Nifedipine in the treatment of migraine headache and amaurosis fugax in patients with systemic lupus erythematosus.
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Headache and amaurosis fugax in an obese woman.
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Young patient with headache and amaurosis fugax.
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[Risk factors in patients with amaurosis fugax].
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BACKGROUND
In the diagnosis of retinal and cerebral vascular occlusion and migraine amaurosis fugax may be an important symptom. To evaluate the relation between cardiovascular diseases and amaurosis fugax we investigated the risk factors in patients with amaurosis fugax.
METHODS
Twenty-four
Amaurosis fugax and ocular infarction in adolescents and young adults.
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Because the cause and natural history of amaurosis fugax and ocular infarction are unknown in most younger patients, we reviewed the records of 83 patients who had become symptomatic before the age of 45. Cerebral transient ischemic attacks had occurred in 9 of these patients but no case of stroke
Amaurosis fugax in teenagers. A migraine variant.
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Sudden, transient loss of vision in one eye (amaurosis fugax) is associated frequently with atherosclerosis of the internal carotid artery in adults and may herald a stroke. Thus, cerebral angiography is often performed. Amaurosis fugax in children is uncommon and an underlying cause is rarely
Headache associated with transient ischemic attacks.
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Sixty (29%) of 205 consecutive patients with transient ischemic attacks registered in a hospital stroke data base had headache within 72 hours of onset. Headache was significantly more common in nonsmokers (odds ratio = 2.8; 95% confidence interval = 6.7 to 1.2). Headache was infrequent in patients
Amaurosis fugax in young people.
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Nine young adults (median age 19.5 years) who suffered from amaurosis fugax (AF) are described. The attacks of AF were short in duration and preceded by premonitory symptoms in five cases and by a migrainous headache in two. In five patients the visual loss progressed in a lacunar pattern unlike the
[A case of giant meningioma that induced visual changes similar to those observed in amaurosis fugax in an early stage of disease].
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The patient was a 47-year-old female. A giant meningioma was detected in the right middle cranial fossa by exploration for the cause of right-sided transient monoocular blindness of a 1-2 minute duration accompanied by headache. The headache was localized on the right side, occurred more frequently
Neuro-Ophthalmic Symptoms of Primary Headache Disorders: Why the Patient With Headache May Present to Neuro-Ophthalmology.
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[Amaurosis fugax with elevated acute phase proteins].
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Case report of a 66-year-old woman with episodes of amaurosis fugax and hemicranic headache with otherwise normal ophthalmologic and neurological examinations and normal imaging. While ESR was in the normal range for patient's age, acute phase proteins (C-reactive protein and fibrinogen) were
Amaurosis fugax under the age of 40 years.
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Sixteen patients who presented under the age of 40 years with amaurosis fugax have been studied. Follow up from the time of presentation was one to 13 years with a median of 3 years. One patient whose attacks of uniocular visual loss were associated with headache developed a permanent uniocular
Temporal arteritis-like presentation of carotid atherosclerosis.
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A 68 year-old woman presented with a two-week history of amaurosis fugax, ipsilateral fronto-temporal headache and jaw claudication suggesting carotid giant cell arteritis. However, this syndrome proved to be due to atherosclerosis causing complete occlusion of the external carotid artery at its
[The role of chronic venous insufficiency in the pathogenesis of brain diseases].
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Recent data from the literature suggest a greater role of chronic venous insufficiency in the pathogenesis of a variety of brain disorders than previously thought. The more perfect method of imaging the structure and brain function contributed to it. The method of choice in the diagnosis of cerebral
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