amylase/atrofia

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Significance of Amylase Monitoring in Peritoneal Drainage Fluid after Splenectomy: A Clinical Analysis of Splenectomy in 167 Patients with Hepatolenticular Degeneration.

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Different kinds of complications after splenectomy in hepatolenticular degeneration patients with hypersplenism have been reported in the past decades, but studies on pancreatic fistula and the corresponding targeted prevention and treatment after splenectomy still remain much unexplored. The

Degeneration secretion of amylase from the parotid gland of the rat.

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In anaesthetized rats the parotid saliva secreted during degeneration of the auriculo-temporal nerve contains amylase in fairly constant concentration, the amylase output varying with the secretory rate. The amount of amylase was not reduced by adrenoceptor blocking agents; it was increased by

Parotid degeneration secretion of amylase in vitro in the rat following sympathectomy.

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After extirpation of the superior cervical ganglion in the rat a degeneration secretion of amylase occurs in an in vitro preparation of the parotid gland. It can be detected about 14 h after the sympathectomy, reaches a maximum after about 17 h and then slowly subsides. It is abolished by atenolol,

Biased mutagenesis in the N-terminal region by degenerate oligonucleotide gene shuffling enhances secretory expression of barley alpha-amylase 2 in yeast.

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Recombinant barley alpha-amylase 1 (rAMY1) and 2 (rAMY2), despite 80% sequence identity, are produced in very different amounts of 1.1 and <0.05 mg/l, respectively, by Saccharomyces cerevisiae strain S150-2B. The low yield of AMY2 practically excludes mutational analysis of structure-function

Secretion of amylase from the rat parotid salivary gland after degeneration of the auriculotemporal nerve.

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The output of amylase into saliva secreted after injection of methacholine or substance P was increased after parasympathetic denervation, but the salivary concentration of amylase was unchanged. The increased output corresponded to the increased flow. Isoprenaline injected during the

A method for inducing exocrine atrophy and collecting juice from the nonatrophied pancreas in the rat.

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A method for the long-term diversion of bile in the rat is described. It was used to induce complete and uniform pancreatic exocrine atrophy, as confirmed by a marked loss of weight of the gland, very low pancreatic tissue amylase, and by histological examination. The technique induces changes in

Comparison of standardized patients with high-fidelity simulators for managing stress and improving performance in clinical deterioration: A mixed methods study.

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BACKGROUND The use of standardized patients in deteriorating patient simulations adds realism that can be valuable for preparing nurse trainees for stress and enhancing their performance during actual patient deterioration. Emotional engagement resulting from increased fidelity can provide

Pancreatic duct obstruction with an acrylate glue: a new method for producing pancreatic exocrine atrophy.

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Ten dogs have been used in a study which demonstrates the feasibility of producing total pancreatic duct obstruction with alkyl-alpha-cyanoacrylate glue. Low pressure injection of glue into the pancreatic duct has led to a reproducible pancreatic atrophy with preservation of the islets. A sharp rise

[The adaptational-compensatory reactions of the digestive system during the development of pancreatic atrophy].

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Adaptive-compensatory processes have been revealed in the digestive system in pancreatic atrophy. These processes promote: a) the compensation of the pancreatic enzymatic insufficiency and of the cavitary digestion decrease by the alpha-amylase adsorbed at the small intestinal epithelium, b) the

[Physiological and biochemical characteristics of changes in pancreatic secretory activity during the development of atrophy in dogs].

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In pancreatic atrophy the normal digestive dynamics of pancreatic juice volume, concentrations and outputs of proteolytic enzymes, alpha-amylase and bicarbonate are disturbed. The changes of enzymic dynamics are revealed before those of pancreatic juice volume and bicarbonate concentration. The

Quantitative assay of Schieff and PAS reactivity during nerve degeneration in rats.

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Quantative histochemical analysis of nerve degeneration in rats from zero to 192 hours was studied utilizing both Schiff reagent and PAS reaction. In addition, amylase digestion prior to PAS staining and aniline blockade of Schiff reactivity were employed. The staining intensity of all the reaction

Sporadic case resembling autosomal-dominant motor system degeneration (Azorean disease complex).

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We describe a case of an adult-onset progressive dystonia with external ophthalmoplegia, occurring in a black man without a family history of neurologic disorders. Neuropathologic examination demonstrated neuronal loss and gliosis in the anterior horn and Clarke's column in the spinal cord, nuclei

Amylase-Trypsin Inhibitors in Wheat and Other Cereals as Potential Activators of the Effects of Nonceliac Gluten Sensitivity.

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Nonceliac gluten sensitivity (NCGS) is a gluten-related gastrointestinal disorder distinct from celiac disease (CD) and gluten allergy that is not easy to diagnose due to the lack of biomarkers. It is characterized by intestinal symptoms and extraintestinal manifestations with the consumption of

Clinical and pathologic findings in two draft horses with progressive muscle atrophy, neuromuscular weakness, and abnormal gait characteristic of shivers syndrome.

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Two Belgian geldings, 4 and 14 years old, respectively, with muscle atrophy, weakness, and abnormal gait characteristic of severe advanced shivers were examined clinically and on necropsy. Neurologic examination revealed no evidence of ataxia, and the clinical diagnosis was neuromuscular weakness

Gibberellin induces alpha-amylase gene in seed coat of Ipomoea nil immature seeds.

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Two full-length cDNAs encoding gibberellin 3-oxidases, InGA3ox1 and InGA3ox2, were cloned from developing seeds of morning glory (Ipomoea nil (Pharbitis nil) Choisy cv. Violet) with degenerate-PCR and RACEs. The RNA-blot analysis for these clones revealed that the InGA3ox2 gene was

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