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angiodysplasia/edema
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A capillary hemostasis mechanism regulated by sympathetic tone and activity via factor VIII or von Willebrand's factor may function as a "capillary gate" and may explain angiodysplasia, angioneurotic edema, and variations in systemic vascular resistance.
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The sympathetic nervous system (SNS) may directly or indirectly govern the release of von Willebrand's factor (VWB), which may be a hormone, from the vascular endothelium, which may be a gland. VWB is known to stabilize the labile VIIIC component of Factor VIII complex, and may thereby determine its
Small intestinal edema had the strongest correlation with portal venous pressure amongst capsule endoscopy findings.
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OBJECTIVE
Previous studies have reported small intestinal lesions in patients with portal hypertensive disease. However, the etiology of these lesions is not clear, as portal venous pressure was not measured in any of these studies. The aim of this study is to clarify the association between small
An imaging evaluation of angiodysplasia syndromes.
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Current imaging techniques such as magnetic resonance, magnetic resonance angiography, computer tomography, ultrasound, plain x-rays, and lymphangioscintigraphy have enhanced the ability to define blood and lymph vascular malformations in more precise pathophysiologic terms. Not only can these
Frontiers in lymphatic microsurgery.
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The Authors report an overview on the modern surgical treatment of peripheral lymphoedema. The aim of lymphatic microsurgical operations is to drain the lymph either toward the venous circulation (lympho-venous shunts) or the lymphatic collectors above the obstacle to the lymph flow, with the
Endoscopic characterization of the small bowel in patients with portal hypertension evaluated by double balloon endoscopy.
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BACKGROUND
The endoscopic abnormalities present in the small bowel (SB) of patients with portal hypertension (PH) are not well understood. This study sought to evaluate endoscopic findings of the SB in patients with PH by double balloon endoscopy (DBE).
METHODS
We evaluated the endoscopic findings
Lethal outcomes in Klippel-Trenaunay syndrome.
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Klippel-Trenaunay syndrome (KTS) is an uncommon congenital angiodysplasia that manifests in infancy and is characterized by venous and lymphatic malformations of the skin, soft tissue, and bone causing limb hypertrophy. We report 2 patients with long-term KTS who developed lethal complications from
Lymphangiogenesis and lymphangiodysplasia: from molecular to clinical lymphology.
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The lymph vascular system parallels the blood vasculature and as one of its key functions returns liquid and solutes to the bloodstream, including macromolecules that have escaped from blood capillaries and entered the interstitium. In conjunction with interspersed lymph nodes and lymphoid organs,
Altered Prostasin (CAP1/Prss8) Expression Favors Inflammation and Tissue Remodeling in DSS-induced Colitis.
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Inflammatory bowel diseases (IBD) including ulcerative colitis and Crohn's disease are diseases with impaired epithelial barrier function. We aimed to investigate whether mutated prostasin and thus, reduced colonic epithelial sodium channel activity predisposes to develop an experimentally dextran
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