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arthus reaction/edema
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Mechanism of action of colchicine. I. Effect of colchicine and its analogs on the reversed passive Arthus reaction and the carrageenan-induced hindpaw edema in the rat.
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Colchicine and N-desacetyl-N-methylcolchicine suppressed both the reversed passive Arthus reaction and the carrageenan-induced edema in the rat. Colchicine, 2-desmethyl-colchicine glucoside and trimethylcolchicine acid had no effect on either model of inflammation. The ability or inability of these
[Arthus reaction in the rat paw. Histological and immunofluorescent studies and their correlation to the edema formation].
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[Studies on anti-ulcerogenic protein in inflamed rabbit skin tissues. II. Isolation of inhibitory substance to gastric juice secretion and carrageenin edema formation from Arthus reaction site (author's transl)].
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[Fibrinolytic activity in the foot pad skin of the rat during direct passive Arthus reaction and pharmacologically induced foot pad edema].
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[Enzyme activities in the edema fluid of rat paw edemas, induced by dextran, formaldehyde and Arthus reaction].
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[On the significance of proteases for the Arthus reaction and pharmacologically induced edema in rat paws].
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[Influence of a potassium depleting diet on rat paw edemas induced by dextran, formalin or an Arthus reaction].
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Role of C-C chemokine receptors 1 and 5 and CCL3/macrophage inflammatory protein-1alpha in the cutaneous Arthus reaction: possible attenuation of their inhibitory effects by compensatory chemokine production.
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The deposition of immune complexes induces an acute inflammatory response with tissue injury. Immune complex-induced tissue injury is mediated by inflammatory cell infiltration that is highly regulated by multiple chemokines. To assess the role of the chemokine receptors CCR1 and CCR5, and a ligand
Effects of cholinotropic and cytostatic drugs on the development of Arthus reaction.
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Antimuscarinic effects of ipratropium bromide and atropine are associated with prevention of the development of Arthus reaction, while the cytostatic effects of cyclophosphamide and doxorubicin lead to involution of the thymus and spleen, suppression of antibody production, and aggravation of
The vaccines-associated Arthus reaction.
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The Arthus reaction is a rare adverse reaction that usually occurs after vaccination with large and more severe local reactions, belonging to type Ⅲ hypersensitivity reaction. This reaction is characterized by pain, swelling, induration (Tissue that becomes firm) and edema, even accompanied by
Immune complexes and inflammation. A study of the activity of anti-inflammatory drugs in the reverse passive arthus reaction in the rat.
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The reverse passive Arthus reaction in rat skin was quantitated by using increase in wet weight as a measure of edema and extractable myeloperoxidase as a measure of the intensity of polymorphonuclear leukocyte (PMN) infiltration. Treatment of the animal with dexamethasone prior to challenge with
Role of P-selectin in the early stage of the Arthus reaction.
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P-selectin is rapidly translocated to the surface of endothelial cells and platelets following exposure to chemical mediators such as histamine, thrombin and complement factors. The Arthus reaction is caused by vascular injury which is initiated by the local deposition of the immune complex followed
Impaired inflammatory responses in the reverse arthus reaction through genetic deletion of the C5a receptor.
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We recently demonstrated that gene-targeted disruption of the C5a anaphylatoxin receptor prevented lung injury in immune complex-mediated inflammation. In this study, we compare the effect of C5aR deficiency in immune complex-induced inflammation in the peritoneal cavity and skin with the results
[Suppressive effects of lanoconazole on arthus phenomenon in vivo and on production and functions of TNF in vitro].
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The anti-inflammatory effect of lanoconazole (LCZ) was investigated in vivo and in vitro. The effect of LCZ was evaluated on the inflammatory reactions elicited by intradermal injection of ovalbumin to ovalbumin-immunized rabbits, as an Arthus phenomenon. A one or two % cream preparation of LCZ was
The Arthus reaction in rodents: species-specific requirement of complement.
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We induced reverse passive Arthus (RPA) reactions in the skin of rodents and found that the contribution of complement to immune complex-mediated inflammation is species specific. Complement was found to be necessary in rats and guinea pigs but not in C57BL/6J mice. In rats, within 4 h after
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