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atherosclerosis/edema

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The prevalence of pedal edema (PE) and its associations with abnormal cardiac structure/function, natriuretic peptides, and incident heart failure (HF) is unknown, especially in community-dwelling adults without a history of cardiovascular disease. Out of 5004 MESA (Multiethnic Study of

Lymphatic and transcapillary forces in patients with edema following operation for lower limb atherosclerosis.

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Intralymphatic end pressure and Starling pressures (interstitial fluid pressure (Pif), plasma and interstitial fluid colloid osmotic pressures (COPpl and COPif)) were measured in leg subcutaneous tissue in 5 patients with local leg edema following femoropopliteal reconstruction for lower limb

Edema in the lower limb following arterial reconstruction for atherosclerosis. A study of pathogenetic mechanisms.

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Determination of edema in porcine coronary arteries by T2 weighted cardiovascular magnetic resonance.

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BACKGROUND Inflammation plays a pivotal role in all stages of atherosclerosis. Since edema is known to be an integral part of inflammation, a noninvasive technique that can identify edema in the coronary artery wall may provide unique information regarding plaque activity. In this study, we aimed to
The development of experimental atherosclerosis in rabbits is accompanied by infiltration into the aorta intima of low density lipoproteins and marked proliferation of smooth muscle cells. The activated smooth muscle cells take part in desmoplastic reactions being the main source of collagen
Introduction: Atherosclerosis is a trigger in the development of cardiovascular disease. Complications of atherosclerosis give reason to search for new criteria, diagnostic concepts, treatment methods and active preventive measures. The aim of our work is to study of the structural
Ultrasound Doppler flowmetry during local exposure to subatmospheric pressure (limb negative pressure exposure--LNPE) was used to study the effect of increased transmural pressure on femoral artery blood flow. Twelve healthy controls and patients with lower limb atherosclerosis--before (n = 11) and

A case of marked hyperlipoprotein(a)emia associated with nephrotic syndrome and advanced atherosclerosis.

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In 1989, we encountered a 68-year-old male patient with marked hyperlipoprotein(a)emia (hyperLp(a)emia), who was being treated for hypertension and arteriosclerotic obliterans (ASO) at an outpatient clinic of our hospital. He began to develop leg edema in 2002 and was referred to the Department of

Traumatic brain injury, coronary atherosclerosis and cardiovascular mortality.

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BACKGROUND Traumatic-brain-injury (TBI) is a devastating-condition resulting in cerebral edema and ischemia. This study investigates the association of mild-TBI (mTBI) to sub-clinical atherosclerosis and cardiovascular (CV) mortality. METHODS Five hundred and forty-three veterans without known

Heart failure in a macaw with atherosclerosis of the aorta and brachiocephalic arteries.

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A female severe macaw (Ara severa) that was at least 11 years old was evaluated for sudden onset of exercise intolerance and dyspnea. Radiography revealed a large heart silhouette, an increase in prominence of the brachiocephalic arteries, and a diffuse increase in opacity of the lungs. Lateral

[Atherosclerosis and the kidney].

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Diffuse atherosclerosis entails a 15-30% risk of plaques on renal arteries (ARAS), with a correlation with coronary atherosclerosis. Ischemia induces generation of angiotensin II (Ang II) that maintains sufficient hydrostatic pressure within the tuft to preserve the GFR. Ang II inhibition suppresses

[Structural transformations of the intima of arteries of the human pia mater in atherosclerosis].

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Using scanning and transmission microscopy, the structural transformations of the internal coating of the pial arteries have been investigated in patients with atherosclerosis (grade I+). The examination has revealed marked changes in the intima expressed in variable degree disorganization of the

Imaging of carotid artery vessel wall edema using T2-weighted cardiovascular magnetic resonance.

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BACKGROUND Atherothrombosis remains a major health problem in the western world, and carotid atherosclerosis is an important contributor to embolic ischemic strokes. It remains a clinical challenge to identify rupture-prone atherosclerotic plaques before clinical events occur. Inflammation,
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