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Observations on lipid composition with particular reference to cardiolipin of rat heart after feeding rapeseed oil.

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The influence of dietary rapeseed oil on the lipid classes and fatty acid pattern of rat heart homogenate and mitochondria has been investigated after feeding a diet with 9.8 weight- % erucic acid for 10 days and 1.4 and 2.6% erucic acid for 28 days. The rats treated with 9.8% erucic acid showed a

Studies on phospholipids with particular reference to cardiolipin of rat heart after feeding rapeseed oil.

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Changes in rat heart phospholipid composition after rapeseed oil feeding.

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The influence of long duration rapeseed oil feeding with high or low levels of erucic acid has been investigated on rat heart phospholipids. The rats treated for 20 wk with rapeseed oil containing 46.2% erucic acid showed a twofold increase in the sphingomyelin content of the heart. Treatment with

Changes in fatty acid composition of cardiac mitochondrial phospholipids in rats fed rapeseed oil.

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Male Wistar rats were fed rapeseed oil containing high or low levels or erucic acid for 20 weeks, and changes in the fatty acid composition of cardiac mitochondrial phospholipids were studied. Treatment with rapeseed oil containing 46.2% erucic acid showed incorporation of 22:1 (5.6%) into isolated

Different dietary fats influence serum and tissue lipids and anti-cardiolipin antibody levels in autoimmune-prone NZB/W F1 mice.

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To investigate the influence of different dietary fats on lipids and anti-cardiolipin antibody levels, autoimmune NZB/W F1 mice were fed on diets containing 200 g dietary fat as palm oil, lard-soyabean oil (1:1, w/w), soyabean oil, rapeseed oil or fish oil/kg. In addition, each dietary fat group was

Rapeseed oil-rich diet alters hepatic mitochondrial membrane lipid composition and disrupts bioenergetics.

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Diet is directly related with physiological alterations occurring at a cell and subcellular level. However, the role of diet manipulation on mitochondrial physiology is still largely unexplored. Aiming at correlating diet with alterations of mitochondrial membrane composition and bioenergetics,

Dynamic modulation of mitochondrial inner-membrane lipids in rat heart by dietary fat.

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A novel longitudinal feeding design was used to investigate the controlling influence of dietary fatty acids on the dynamic incorporation of fatty-acyl chains into phosphatidylcholine, phosphatidylethanolamine and cardiolipin in inner membrane of cardiac mitochondria. Rats were fed a

[Variations in the lipid and fatty acid composition of cardiac tissue during perfusion of hearts isolated from rats on a high colza oil diet].

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After perfusion, during one hour, of isolated working hearts extracted from Rats fed (60 days) with diet containing 15% of rapeseed oil, we have found a decrease of triglycerides and phosphatidic-cardiolipin fractions, particularly of fractions containing erucic acid. In contrast with this finding,

The effects of partially hydrogenated marine oils on the mitochondrial function and membrane phospholipid fatty acids in rat heart.

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The influence of dietary partially hydrogenated marine oils containing docosenoic acid on rat heart mitochondrial membrane phospholipid fatty acid composition was studied with particular reference to cardiolipin and oxidative phosphorylation. Five groups of male weanling rats were fed diets

Mitochondrial-membrane polar-head-group composition is influenced by diet fat.

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Male Sprague-Dawley rats were fed diets containing 20% (w/w) soya-bean oil, high-erucic acid rapeseed oil or low-erucic acid rapeseed oil for 0, 12 or 23 days. The type of fat present in the diet had no effect on the total phospholipid content of heart mitochondria (micrograms/mg of protein) but did

Dietary fatty acids affect mitochondrial phospholipid compositions and mitochondrial gene expression of rainbow trout liver at different ages.

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Mitochondria are among the first responders to various stressors that challenge the homeostasis of cells and organisms. Mitochondrial decay is generally associated with impairment in the organelle bioenergetics function and increased oxidative stress, and it appears that deterioration of

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