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Página 1 a partir de 42 resultados
Role of nitric oxide in catalepsy and hyperthermia in morphine-dependent rats.
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The possible involvement of nitric oxide (NO) in morphine-induced catalepsy and hyperthermia was studied in morphine-dependent rats. Four days repeated injection regimen was used to induce morphine dependence, which was assessed by naloxone challenge (0.5 mg x kg(-1), s.c.). Pretreatment of rats
beta-Endorphin: central sites of analgesia, catalepsy and body temperature changes in rats.
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beta-Endorphin was microinjected into rat brain in order to localize central sites associated with some of its pharmacologic effects: namely, analgesia (inhibition of the tail-flick response), catalepsy and changes in body temperature. Microinjections (1 microliter) were made bilaterally under
(RS)-alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid: wet dog shakes, catalepsy and body temperature changes in rats.
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(RS)-alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) was microinjected into the lateral brain ventricle of conscious rats in order to evaluate its pharmacological effects. Microinjection (5 microliter) were made unilaterally and the effects of AMPA were assessed for 6 hr. AMPA
Skeletal muscle thermogenesis: its role in the hyperthermia of conscious rats given morphine or beta-endorphin.
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The role of skeletal muscle thermogenesis (increases in skeletal muscle tone) in the hyperthermic responses of conscious, unrestrained rats given acute or repeated i.p. or i.c.v. injections of morphine sulfate (MS) or beta-endorphin was investigated. Initial blood gas experiments showed that rats
[A young woman with a labile mood, hyperactivity, hyperthermia and exhaustion: symptoms of lethal catatonia].
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A 22-year-old woman went through a period with a labile mood that first turned into psychotic hyperactivity and was then followed by hyperthermia and exhaustion. This was accompanied by diminished consciousness, sinus arrhythmia and respiratory insufficiency. With a working diagnosis of 'lethal
Catatonia causing permanent cognitive impairment: a case study.
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OBJECTIVE
We describe a case of psychogenic malignant catatonia resulting in permanent cognitive impairment.
BACKGROUND
Catatonia is a neuropsychiatric syndrome characterized by catalepsy, negativism, mutism, muscular rigidity, and mannerisms, often accompanied by autonomic instability and fever.
Malignant catatonia with severe bronchorrhea and its response to electroconvulsive therapy.
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A 21-year-old female presented excitement, auditory hallucination, monologue, and insomnia. After 1 week of risperidone administration, she showed hyperthermia, salivation, and muscle rigidity. Risperidone was discontinued, but stupor, convulsions, and respiratory distress developed. In the
C-fragment of lipotropin--an endogenous potent analgesic peptide.
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1 A series of peptides derived from porcine lipotropin was examined for analgesic and other morphine-like properties on infusion into the cannulated third ventricle of cats.2 Lipotropin (LPH 1-91) itself produced no analgesia or other morphine-like effects when infused in a dose of 150 mug.3
[Some pharmacologic comments on the action of glycine derivatives in relation to peculiarities of the central glycinergic system].
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The antagonism of the effect of reserpine on the thermoregulation in rats, the potentiation of the reserpine catalepsy with the simultaneous increase of the irritability of rats, the hyperthermia in mice, etc., obtained with some new glycine derivatives, are discussed in relation with recent data on
SR 95191, a selective inhibitor of type A monoamine oxidase with dopaminergic properties. I. Psychopharmacological profile in rodents.
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SR 95191 [3-(2-morpholino-ethyl-amino)-4-cyano-6-phenyl-pyridazine], a novel compound, has been shown in preliminary experiments to inhibit type A monoamine oxidase (MAO). This report describes the activities of SR 95191 in behavioral experiments in mice and rats and shows that SR 95191 has the
The central action of salbutamol, a beta-agonist with a potential antidepressant activity.
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The pharmacological profile of salbutamol, an agonist of beta-adrenergic receptors and a potential antidepressant drug, and its effect on the central serotonin system were studied. It was found that salbutamol either had no effect, or, at higher doses, inhibited the spontaneous activity of mice and
Central antiserotonergic and antidopaminergic action of pirenperone, a putative 5-HT2 receptor antagonist.
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Pirenperone, an antagonist of 5-HT2 but not 5-HT1 receptors, has been studied for its central antiserotonergic and antidopaminergic activity. Pirenperone (0.00525-0.1 mg/kg) antagonized dose-dependently stimulation of the hind limb flexor reflex in spinal rats induced by LSD, quipazine or
Central action of glaucine in rodents.
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It was found that glaucine (7, 14 and 28 mg/kg ip) decreased locomotor activity, did not affect the body temperature and potentiated the amphetamine and apomorphine stereotypy but did not affect the amphetamine hypermotility. Besides, glaucine antagonized haloperidol-induced catalepsy of the rats.
In vitro opioid receptor affinity and in vivo behavioral studies of Nelumbo nucifera flower.
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BACKGROUND
Nelumbo nucifera Geartn., known as sacred lotus, has been used traditionally in South East Asia as a traditional medicine for various CNS disorders including stress, fever, depression, insomnia, and cognitive conditions.
OBJECTIVE
To investigate the in vitro cannabinoid and opioid
Neuropharmacological studies on Panax ginseng.
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Panax ginseng root powder is extensively used in the Far East for a wide variety of clinical ailments and to improve general physical and mental wellbeing. It is now also being used in the Occident because of the adaptogenic activity of the plant. The present investigation was conducted to evaluate
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