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dopa/hemorragia
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Holmes tremor secondary to brainstem hemorrhage responsive to levodopa: report of 2 cases.
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We report 2 patients who presented a brainstem hemorrhage and who, after 1 and 6 months, respectively, developed a 4-Hz postural and resting tremor consistent with Holmes tremor, which severely interfered with the activities of daily living. In both cases, levodopa dramatically improved the tremor.
Levodopa-responsive parkinsonism after aneurysmal subarachnoid hemorrhage.
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BACKGROUND
Levodopa-responsive Parkinsonism is a rare complication of subarachnoid hemorrhage and no cases have been reported to occur in the absence of hydrocephalus.
METHODS
Case report.
RESULTS
We describe a 42-year-old woman who developed progressive severe Parkinsonism in the second week after
Holmes tremor after brainstem hemorrhage, treated with levodopa.
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Holmes tremor is a rare movement phenomenon, with atypical low-frequency tremor at rest and when changing postures, often related to brainstem pathology. We report a 70-year-old female patient who was presented with dystonic head and upper limb tremor after brainstem hemorrhage. The patient had
Postoperative bleeding associated with levodopa therapy.
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Postmenopausal bleeding: another side-effect of levodopa.
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Levodopa-responsive Parkinsonism following bilateral putaminal hemorrhages.
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Levodopa-responsive Holmes head titubation caused by midbrain cavernoma hemorrhage
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Midbrain tremor and hypertrophic olivary degeneration after pontine hemorrhage.
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A severe rest tremor arose in a patient's right arm 9 months after a pontine tegmental hemorrhage. Magnetic resonance studies at 4 and 10 months showed residual hemosiderin in the pons and increasing hypertrophic olivary degeneration (HOD) affecting primarily the left olive. The tremor was
Gastrointestinal bleeding and massive liver damage in neuroleptic malignant syndrome.
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BACKGROUND
Neuroleptic malignant syndrome (NMS) is a rare side effect of antipsychotic therapy characterized by fever, muscular rigidity, altered mental status, increased level of serum creatinine phosphokinase, and increased number of white blood cells. The mortality rate of patients with NMS
Levodopa therapy of patients with Parkinsonism and heart disease.
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Forty patients with Parkinsonism and heart disease were studied before and during the administration of levodopa. Patients with increasing angina, myocardial infarction within the previous year, pre-existing severe postural hypotension, or transient cerebral ischaemia were excluded. Thirty-eight
Serotonin Syndrome Following Combined Administration of Dopaminergic and Noradrenergic Agents in a Patient With Akinetic Mutism After Frontal Intracerebral Hemorrhage: A Case Report.
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BACKGROUND
Serotonin syndrome (SS) is a potentially life-threatening condition that can be caused by use of proserotonergic drugs. Several studies have reported that combined administration of various medications may induce SS. We report a case of SS in a patient who was being treated with
Parkinsonism after external ventricular drainage in a patient with intraventricular hemorrhage.
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We report a patient who presented with Parkinsonism after external ventricular drainage (EVD) for an intraventricular hemorrhage (IVH). We also demonstrate dopaminergic system dysfunction using (18)F-florinated-N-3-fluoropropyl-2-β-carboxymethoxy-3-β-(4-lodophenyl) nortropane ((18)F-FP-CIT) positron
Successful Management of Hemorrhage-Associated Hemiballism After Subthalamic Nucleus Deep Brain Stimulation with Pallidal Stimulation: a Case Report.
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BACKGROUND
Deep brain stimulation has been widely used for treating several movement disorders including idiopathic Parkinson disease (IPD). The development of hemiballism after an iatrogenic injury to the subthalamic nucleus (STN) such as postoperative hemorrhage or stroke is rare. Employing
Treatment of post-traumatic midbrain resting-kinetic tremor with combined levodopa/carbidopa and carbamazepine.
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A patient with a post-traumatic midbrain haemorrhagic lesion documented by magnetic resonance imaging (MRI) presented with a combined resting-kinetic contralateral upper extremity tremor. The resting tremor component responded to levodopa/carbidopa, while the kinetic component improved with the
Interaction between levodopa and enteral nutrition.
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OBJECTIVE
To report and discuss a drug-nutrient interaction involving levodopa and protein in enteral nutrition.
METHODS
A 77-year-old male with Parkinson's disease was admitted to an intensive care unit for an intracerebral hemorrhage. To provide nutritional support, an oral gastric tube was placed
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