fructose intolerance/triglyceride

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Patients with aldolase B deficiency are characterized by an increased intrahepatic triglyceride content.

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There is an ongoing debate on whether and how fructose is involved in the pathogenesis of nonalcoholic fatty liver disease (NAFLD). A recent experimental study showed an increased intrahepatic triglycerides (IHTG) content in mice deficient for aldolase B (aldo B-/-), the enzyme that

Are heterozygous carriers for hereditary fructose intolerance predisposed to metabolic disturbances when exposed to fructose?

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UNASSIGNED High fructose intake causes hepatic insulin resistance and increases postprandial blood glucose, lactate, triglyceride, and uric acid concentrations. Uric acid may contribute to insulin resistance and dyslipidemia in the general population. In patients with hereditary fructose

[Fructose and fructose intolerance].

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Although fructose was discovered in 1794, it was realised in recent decades only that its malabsorption can lead to intestinal symptoms while its excessive consumption induces metabolic disturbances. Fructose is a monosaccharide found naturally in most fruits and vegetables. Dietary intake of

Kidney and vascular function in adult patients with hereditary fructose intolerance

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Objective: Previous studies have shown that patients with hereditary fructose intolerance (HFI) are characterized by a greater intrahepatic triglyceride content, despite a fructose-restricted diet. The present study aimed to examine the long-term consequences of HFI on other

Comparative effects of some carbohydrates on serum sugars, triglycerides and digestive hydrolases.

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For 3 weeks, rats were fed diets containing 60 p. 100 carbohydrate in the form of starch (wheat flour), purified sucrose, commercial sugar or a commercial sweetener containing a mixture of glucose and fructose. Glycemia was lower during the day than at night, and it was lowest in the starch-fed

Abdominal ultrasonography in inheredited diseases of carbohydrate metabolism.

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OBJECTIVE To determine the usefulness of abdominal sonography in inherited diseases of carbohydrate metabolism. METHODS Thirty patients (age range, 4 months to 27 years) with glycogen storage diseases, galactosemia, disorders of fructose metabolism were studied with sonography. Echogenicity of the

Nutritional considerations and management of the child with liver disease.

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Nutritional management of the infant and child with liver disease is highly dependent upon the type of liver disease. Acute liver disease, such as that secondary to viral hepatitis, requires no specific nutritional therapy with the exception that branched-chain amino acid supplements may be

Triose Kinase Controls the Lipogenic Potential of Fructose and Dietary Tolerance

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The surge in fructose consumption is a major factor behind the rapid rise of nonalcoholic fatty liver disease in modern society. Through flux and genetic analyses, we demonstrate that fructose is catabolized at a much higher rate than glucose, and triose kinase (TK) couples fructolysis with

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