muscle weakness/necrose

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Respiratory and limb muscle weakness induced by tumor necrosis factor-alpha: involvement of muscle myofilaments.

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The respiratory and limb skeletal muscles become weakened in sepsis, congestive heart failure, and other inflammatory diseases. A potential mediator of muscle weakness is tumor necrosis factor (TNF)-alpha, a cytokine that can stimulate muscle wasting and also can induce contractile dysfunction

[A case with severe respiratory muscle weakness due to chronic myositis associated with PBC].

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We report a 37-year-old woman with slowly developing muscular weakness for 2 years following insidious stiffness of calf muscle. Serum CK was elevated up to 4,207 IU/l. She presented sleepiness, weakness of proximal and truncal muscles and systemic muscular atrophy. While she had not experienced

[Pathophysiology in muscle fiber necrosis and regeneration with a particular reference to regenerating process].

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It is well known that muscle fiber is capable of regeneration after myonecrosis even in a case of muscular dystrophies. We first examined how and when the regulatory genes for myogenesis, MyoD and myogenin were expressed in experimentally induced myonecrosis. MyoD, a responsible gene for myoblast

Acute lindane poisoning with development of muscle necrosis.

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A 35-year-old man ingested food contaminated with lindane, an insecticide containing almost pure gamma hexachlorocyclohexane. Grand mal seizures and severe acidemia developed rapidly. The seizures recurred for nearly 2 hours, then ceased. In addition, the patient had muscle weakness and pain,

[Bitemporal scalp necrosis : a very rare manifestation of giant cell arteritis].

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A 71-year-old woman developed progressive spreading of bitemporal scalp necrosis within 4 weeks accompanied by headaches, myalgia of the shoulder girdle and muscle weakness that had started a few months previously. No additional diseases were reported. The suspected temporal giant cell arteritis

Necrosis of muscle with carcinoma: myositis or myopathy?

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A man aged 59 years developed a rapidly progressive muscular weakness which within a few weeks involved almost the entire musculature. A carcinoma of the stomach was discovered at necropsy. The skeletal muscles showed widespread necrosis with minimal inflammatory reaction. The findings are compared

Spontaneous Progressive Muscle Weakness with Persistent Leukocytosis.

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Iliacus compartment syndrome is a rare clinical condition which can result in a severe, unilateral, femoral neuropraxia. Recognition of this syndrome as the cause of a developing neuropathy is often delayed given a lack of familiarity with this clinical diagnosis and the retroperitoneal location of

Factors related to hip muscle weakness following fixation of acetabular fractures.

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Two hundred twenty-five acetabular fractures treated surgically by one surgeon using a single surgical approach were reviewed to identify the incidence of clinically significant hip muscle weakness. Clinical grade of fair or poor and presence of radiographic signs of osteoarthritis were most

[A case of sarcoid myopathy presenting muscle pain and muscle weakness and with muscle MRI abnormality].

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We experienced a 70-year-old female diagnosed as sarcoidosis. She complained bilateral femoral pain from 70-year-old. 3 months after the onset, she developed muscle weakness extending to her upper extremities with high fever of 38-39 degrees C. The erythema appeared at the right femoral region 4

An in vivo and in vitro H-magnetic resonance spectroscopy study of mdx mouse brain: abnormal development or neural necrosis?

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Duchenne muscular dystrophy (DMD) is an X-linked genetic disorder primarily affecting young boys, often causing mental retardation in addition to the well-known progressive muscular weakness. Normal dystrophin expression is lacking in skeletal muscle and the central nervous system (CNS) of both DMD

Tumour necrosis factor inhibitor-induced myositis in a patient with ulcerative colitis

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Tumour necrosis factor (TNF) inhibitors are known to induce autoimmune diseases, such as lupus-like syndrome; in rare cases, TNF inhibitor-induced myositis has been reported. This report documents the case of a male patient with ulcerative colitis (UC) complicated by TNF inhibitor-induced myositis.

Effect of tumor necrosis factor alpha on electrically induced calcium transients elicited in C2C12 skeletal myotubes.

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Diseases involving chronic inflammation can lead to prolonged exposure of skeletal muscle to inflammatory cytokines such as tumor necrosis factor alpha (TNFalpha), which may contribute to the skeletal muscle weakness seen in these conditions. In this study we examined the effect of a prolonged

Tumor necrosis factor-alpha expression in uvular tissues differs between snorers and apneic patients.

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BACKGROUND Inflammatory changes such as subepithelial edema and excessive inflammatory cell infiltration have been observed in uvular tissues of obstructive sleep apnea (OSA) subjects. The levels of proinflammatory cytokines such as tumor necrosis factor (TNF)-alpha and interleukin-6 are elevated in

[Cutaneous necrosis is predictive of cancer in adult dermatomyositis].

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BACKGROUND Adult dermatomyositis is associated with cancer in 15 p. 100 to 50 p. 100 of cases and, hence, investigations should be systematically performed to search for cancer. A number of predictive factors have been reported. The aim of our study was to search for predictive factors of cancer,

Tumor necrosis factor inhibitor-associated dermatomyositis.

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BACKGROUND Dermatomyositis is an autoimmune disease of unknown etiology characterized by inflammation of the skin and muscles. Several medications have been implicated in the development of dermatomyositis; however, the disease has rarely been linked to the use of tumor necrosis factor (TNF)

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