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nephrosis/hypoxia

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ACUTE TUBULAR NEPHROSIS IN THE NEWBORN INFANT, A MANIFESTATION OF ANOXIA.

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Lower-nephron nephrosis following acute hypoxia. Case report.

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HIF-1alpha expression follows microvascular loss in advanced murine adriamycin nephrosis.

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Cellular hypoxia has been proposed as a major factor in the pathogenesis of chronic renal injury, yet to date there has been no direct evidence to support its importance. Therefore, we examined cortical hypoxia in an animal model of chronic renal injury (murine adriamycin nephrosis; AN) by assessing

Erythropoietin metabolism and pharmacokinetics in experimental nephrosis.

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We studied erythropoietin (EPO) metabolism, regulation, and pharmacokinetics in rats with nephrotic syndrome. Sprague-Dawley rats were randomized into nephrotic (puromycin-induced) and pair-fed control groups. Animals were studied at baseline and after induction of anemia or exposure to hypobaric

Pathogenesis of renal lesions in haemoglobinaemic and non-haemoglobinaemic leptospirosis.

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Hamsters were infected with Leptospira interrogans serovar ballum or Leptospira interrogans serovar pomona and the kidney lesions were compared by light and electron microscopy. Ballum and pomona both caused severe clinical signs and death within 6 days in some animals, although only ballum was

Electrolyte disturbances in foals with severe rhabdomyolysis.

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Marked electrolyte abnormalities characterized by profound hyperkalemia, hyponatremia, hypocalcemia, and hyperphosphatemia were noted in 4 neonatal foals with acute rhabdomyolysis and pigmenturia. In 2 foals, rhabdomyolysis developed 4-6 days after admission for dysmaturity, and in 2 foals,

Expression and regulation of adrenomedullin in renal glomerular podocytes.

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Adrenomedullin (AM) is postulated to exert organ-protective effects. It is expressed in the renal glomeruli, but its roles in the glomerular podocytes have been poorly elucidated. In the present study, we investigated the expression and regulation of AM in recently established conditionally

Deaths: leading causes for 2002.

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OBJECTIVE This report presents final 2002 data on the 10 leading causes of death in the United States by age, race, sex, and Hispanic origin. Leading causes of infant, neonatal, and postneonatal death are also presented. This report supplements the annual report of final mortality

Deaths: leading causes for 2000.

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OBJECTIVE This report presents final 2000 data on the 10 leading causes of death in the United States by age, race, sex, and Hispanic origin. Leading causes of infant, neonatal, and postneonatal death are also presented. This report supplements the annual report of final mortality

Deaths: leading causes for 2001.

Apenas usuários registrados podem traduzir artigos
Entrar Inscrever-se
OBJECTIVE This report presents final 2001 data on the 10 leading causes of death in the United States by age, race, sex, and Hispanic origin. Leading causes of infant, neonatal, and postneonatal death are also presented. This report supplements the annual report of final mortality
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