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[Treatment of chronic recurring parotitis with the protease inhibitor aprotinin (trasylol)].
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Excessive release of glandular kallikrein into the interstitial space around the salivary gland ducts plays a significant role in chronic recurrent parotitis. In the present study 26 patients suffering from acute exacerbated chronic recurrent parotitis were subjected to treatment with the kallikrein
Chronic parotitis: not another SPINKosis.
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BACKGROUND
Pancreatitis and parotitis share several etiological, pathohistological and functional similarities. It arose from recent pancreatitis research that some cases of chronic pancreatitis are associated with mutations of the serine protease inhibitor, Kazal type-1 (SPINK1). We tested the
[Effect of a number of enzyme preparations on the course of experimental parotitis].
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Increase in the weight of rat parotid glands, decrease in the protein concentration and the activity of alpha-amylase with simultaneous activation of the proteolytic enzymes (caseinolytic activity at pH 7.6, activity of cathepsins at pH 5.5) were observed in pyo-traumatic parotiditis. Local
[Effect of the kallikrein-like enzyme salivain on the course of experimental acute parotitis].
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Mechanical trauma of rat parotid gland led to an increase in the size of the gland (due to edema) with simultaneous activation of unspecific proteases, which were estimated at pH 7.6 using casein as a substrate. Administration of kallikrein-like salivary enzyme salivain into non-traumatized parotid
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