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placental insufficiency/albumina

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Perfusion of the intervillous space of the human placenta measured with 99mtechnetium labelled human serum albumin.

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The perfusion of the intervillous space of the human placenta was investigated with 99Tc-Human Serum Albumin and external counting in 45 pregnant women between the 34th and 38th week of gestation. The pregnancy was normal in 34 women, while there was placental insufficiency in 11. Two variables were

Cysteinylation of maternal plasma albumin and its association with intrauterine growth restriction.

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OBJECTIVE We investigated cysteinylation of maternal plasma albumin in an observational study of intrauterine growth restriction (IUGR). High-risk pregnancies and uteroplacental insufficiency (UPI) have been associated with elevated levels of homocysteine, and, in oxidizing environments,
Pregnancy protein beta-1-glycoprotein levels were determined in serum obtained from 100 women with undisturbed pregnancy and 35 women with high risk pregnancy. Concentrations lower than normal, i.e. below the 1s range, were found only in preeclampsia and in pregnancies with intrauterine growth

Food-induced acute generalized exanthematous pustulosis in a pregnant woman.

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A 21-year-old woman of Romany origin, in the third trimester of her fourth pregnancy, was admitted to the hospital because of a generalized erythematous and pustular eruption and desquamation involving her face, neck, trunk, and extremities. The skin changes were accompanied by fever (100.4 degrees
A 38-year-old pregnant woman (19th week of pregnancy) complained of fatigue, cold inducible paresthesias, generalized edema and mild arterial hypertension. Her past medical history was notable for frequent episodes of polyarthralgia and positivity for rheumatoid factor. On admission, acanthocyturia

[Hyperoncotic hemodilution in the ewe asa model of therapy in EPH gestosis (author's transl)].

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Hyperoncotic hemodilution is proposed as a new therapeutic procedure in EPH-gestosis for improvement of uterine blood flow. By infusion of human albumin solutions in acute experiments on pregnant Merino sheep the effects of altered maternofetal colloid osmotic pressure gradients were investigated.

[Study on placental blood flow in late pregnancy by intravenous 99mTc method].

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A method for the continuous recording of uteroplacental blood flow (PBF) in late pregnancies by using 99mTc-albumin has been described. The PBF curve of toxemia of pregnancy has been plotted to indicate small artery spasm in proving ischemic necrosis of placenta. In the PBF of placental
BACKGROUND Maternal perception of reduced fetal movement (RFM) is associated with increased risk of stillbirth and fetal growth restriction (FGR). RFM is thought to represent fetal compensation to conserve energy due to insufficient oxygen and nutrient transfer resulting from placental

Effects of Antenatal Melatonin Treatment on the Cerebral Vasculature in an Ovine Model of Fetal Growth Restriction.

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Chronic moderate hypoxia, such as occurs in fetal growth restriction (FGR) during gestation, compromises the blood-brain barrier (BBB) and results in structural abnormalities of the cerebral vasculature. We have previously determined the neuroprotective and antioxidant effects of maternal

Cerebrovascular adaptations to chronic hypoxia in the growth restricted lamb.

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Chronic moderate hypoxia induces angiogenic adaptation in the brain, reflecting a modulatory role for oxygen in determining cerebrovascular development. Chronic intrauterine fetal hypoxia, such as occurs in intrauterine growth restriction (IUGR) is likely to lead to a reduction in oxygen delivery to

Fetal Growth Restriction Alters Cerebellar Development in Fetal and Neonatal Sheep.

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Fetal growth restriction (FGR) complicates 5-10% of pregnancies and is associated with increased risks of perinatal morbidity and mortality. The development of cerebellar neuropathology in utero, in response to chronic fetal hypoxia, and over the period of high risk for preterm birth, has not
The blood serum levels of glucose, hemoglobin, insulin, cortisol, albumin, alpha-fetoprotein, alpha 2-macroglobulin f and s, alpha 2-antitrypsin inhibitor and alpha 1-protease inhibitor were determined at birth in 5 clinically and morphologically identified mortality groups of pigs. These were
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