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pygeum/isquemia

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Low-dose tadenan protects the rabbit bladder from bilateral ischemia/ reperfusion-induced contractile dysfunction.

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Recent studies indicate that focal ischemia/reperfusion (I/R) can cause the contractile dysfunctions induced in animal models of partial bladder outlet obstruction. Tadenan (Pygeum africanum) pretreatment can prevent the rabbit bladder from developing the contractile and biochemical dysfunctions
Recent studies indicate that localized (regional) ischemia may play an important role in the etiology of the contractile dysfunctions secondary to partial outlet obstruction. Pretreatment with Tadenan® (Pygeum africanum extract) has been shown to protect the rabbit bladder against the development of

Cellular and molecular aspects of bladder hypertrophy.

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Bladder dysfunction secondary to benign prostatic hyperplasia (BPH) is a major affliction associated with ageing. As the disease slowly progresses, the bladder changes from a state of compensation to decompensation, in which there are severe, irreversible alterations in bladder function. Using a

Effect of oral Tadenan treatment on rabbit bladder structure and function after partial outlet obstruction.

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OBJECTIVE There is increasing evidence that the progressive dysfunction induced by partial outlet obstruction is mediated by ischemia-reperfusion, and bladder decompensation results from ischemia-reperfusion induced damage to the cellular and subcellular organelle membranes of nerve and smooth
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