renal colic/fadiga

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Analgesic nephropathy: an underestimated cause of end-stage renal disease.

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Addiction and abuse of antipyretic analgesics has been recognized early after the turn of this century. The incidence markedly increased and the syndrome spread over many countries in the first half of the 20th century. The syndrome and its pathology, consisting of renal papillary necrosis and

Incidental adrenocortical oncocytoma.

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A 66-year-old man with a history of hypertension and poor compliance with therapeutic regimens for the past 10 years presented with right renal colic. Sonographic evaluation of the kidneys revealed an incidental left renal upper pole mass. There were no palpitations, headache, weakness or fatigue,

Primary hyperparathyroidism and Klinefelter's syndrome in a young man.

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We report the association of primary hyperparathyroidism (PHPT) and Klinefelter's syndrome (KS) in a 22-year-old male complaining of worsening fatigue. PHPT was asymptomatic at the diagnosis, but the patient had worsening hypercalcemia and osteoporosis, and developed acute renal colic. He then

Use of Cuban recombinant human erythropoietin in Parkinson's disease treatment.

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BACKGROUND Recombinant human erythropoietin is used primarily to treat anemia. There is evidence of its neuroprotective capacity from preclinical studies in Parkinson's disease and other neurodegenerative diseases. Recombinant human erythropoietin produced in Cuba (ior-EPOCIM) is registered and

Tumors of the parathyroid glands. Changes in clinical features and in noninvasive localization studies sensitivity.

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Primary hyperparathyroidism is the most common cause of hypercalcemia and 80-85% of the patients have parathyroid tumors. The purpose of this retrospective review was to analyse whether differences exist between patients with parathyroid tumors treated in the 1980s and 1990s. Between 1980-1997, 253

Adult onset idiopathic phosphate diabetes.

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This report describes the clinical and biologic data and bone density measurements in 19 adults seen in a rheumatology department, with phosphate diabetes defined by low serum phosphate levels and decreased tubular reabsorption of phosphate in the absence of known etiology. There were 14 males and 5

Adult-onset idiopathic phosphate diabetes. I. Chronic pseudoinflammatory back pain and osteopenia.

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OBJECTIVE to investigate clinical, laboratory test, and bone mineral density abnormalities in 19 adults with phosphate diabetes of unknown etiology diagnosed in a rheumatology department on the basis of a maximal rate for tubular reabsorption of phosphate (TmPO4/GFR) of 0.77 or less. RESULTS there

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