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A one year prospective study of neurogenic stuttering following stroke: incidence and co-occurring disorders.

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In this prospective study, data on incidence, stuttering characteristics, co-occurring speech disorders, and recovery of neurogenic stuttering in a large sample of stroke participants were assessed. Following stroke onset, 17 of 319 participants (5.3%; 95% CI, 3.2-8.3) met the criteria for

Stroke-associated stuttering.

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OBJECTIVE To present patients with stuttering speech in association with stroke. METHODS Case series with follow-up for 5 years, or until the stuttering resolved. METHODS University and community hospital neurology wards, and ambulatory neurology clinics. METHODS Four patients who developed

Remission of life-long stammering after posterior circulation stroke.

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Developmental stammering is relatively common in the adult population. Stammering has a poor prognosis when it persists beyond adolescence and spontaneous or treatment-induced remission is very rare in adults. In this communication we report a case of life-long developmental stammering that resolved

Acquired stuttering after a second stroke in a two-year-old.

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An almost two-year-old left-handed girl with a history of a left-hemisphere stroke at the age of one year developed a transient stutter with newly acquired white matter infarctions. Her course suggests that developmental stuttering may reflect anomalous dominance and/or atypical interhemispheric

X-linked Charcot-Marie-Tooth Disease Presenting with Stuttering Stroke-like Symptoms.

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X-linked Charcot-Marie-Tooth disease (CMTX1) is the second most common form of Charcot-Marie-Tooth disease (CMT). It is caused by a mutation in the gap junction β 1 (GJB1) gene, which encodes for connexin-32. In addition to the peripheral neuropathy and foot deformities observed in classic

A crucial role for the cortico-striato-cortical loop in the pathogenesis of stroke-related neurogenic stuttering.

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Neurogenic stuttering is an acquired speech disorder characterized by the occurrence of stuttering-like dysfluencies following brain damage. Because the onset of stuttering in these patients is associated with brain lesions, this condition provides a unique opportunity to study the neural processes

Return of stuttering after stroke.

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The pathophysiology of developmental or acquired stuttering still remains an enigma. In a few cases, the developmental stuttering that had disappeared spontaneously or as a result of therapy reoccurred following a brain lesion. We report on a patient with return of developmental stuttering following

Neurogenic stuttering as a manifestation of stroke and a mask of dysphonia.

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R. L. was a 52-year-old man who was referred for an SLP consultation to determine the nature of his fluency disorder, whether or not treatment would be beneficial, and finally whether resumption of pre-trauma vocational status was feasible. The patient was involved in a motor vehicle accident with

Rapid resolution of diffusion weighted MRI abnormality in a patient with a stuttering stroke.

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We report the unusually rapid and spontaneous normalisation of low diffusivity that accompanied resolution of acute neurological deficits in a stroke patient who underwent two magnetic resonance imaging examinations within 24 h of symptom onset. Diffusion weighted imaging obtained within hours of

Stuttering as a manifestation of stroke.

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Two cases are described of stuttering as a manifestation of cerebral ischaemia in previously stutter-free individuals. In the first case, stuttering suddenly ceased after carotid endarterectomy; the second patient developed stuttering after a period of three days of complete aphasia after which the

Stroke-associated acquired stuttering.

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In this case report, the syndrome of acquired stuttering is described including its etiology, common presenting features, and differences from developmental stuttering. A case is described in an elderly female patient with sudden acquired stuttering associated with ischemic infarct near the left

Stuttering as a manifestation of right-hemispheric subcortical stroke.

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A right-handed man developed stuttering, without aphasia, as a result of a circumscribed subcortical infarction in the right hemisphere. He never stuttered before and has no family history of stuttering. Damage to callosal pathways coordinating the activity of both hemispheres during speech is

Stuttering due to ischemic stroke.

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Acquired stuttering is a disorder of the fluency of speech. The mechanism underlying stuttering is unknown. It may occur after bilateral and unilateral cortical or subcortical brain damage. We report two cases who had stuttering resulting from left parietal infarction.

Can we stop the stuttering in stroke? Interventions in 40 patients with acute lacunes.

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Whether any treatment can stop fluctuations of stuttering lacunar syndromes (SLS) is unclear. Case reports have variably suggested effectiveness of intravenous thrombolysis, dual antiplatelet treatment, blood pressure augmentation and anticoagulation. We aim to describe our experience

Stuttering pituitary apoplexy resembling meningitis.

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