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tetrandrine/hemorragia
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The effects of acupuncture and traditional Chinese medicines on apoptosis of brain tissue in a rat intracerebral hemorrhage model.
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OBJECTIVE
This study aimed to evaluate the effects of Chinese herbs and acupuncture on the neuronal apoptosis and the expression of apoptosis-related genes in the brain tissue of rats following intracerebral hemorrhage (ICH).
METHODS
Collagenase VII was injected into the caudate nucleus of
Haemodynamic effects of chronic octreotide and tetrandrine administration in portal hypertensive rats.
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Octreotide is an effective portal hypotensive drug in the control of variceal bleeding. Tetrandrine is a type of calcium channel blocker recently reported to reduce portal hypertension. The present study was undertaken to investigate the haemodynamic effects of octreotide and tetrandrine, alone and
Pulmonary toxicity and metabolic activation of tetrandrine in CD-1 mice.
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Tetrandrine, a bisbenzylisoquinoline alkaloid, has demonstrated promising pharmacologic activities. The alkaloid has a great potential for clinical use, so a careful, thorough toxicity evaluation of the alkaloid is required. In the present study, 24 h acute toxicity of tetrandrine was evaluated in
Calcium channel blockers in cirrhotic patients with portal hypertension.
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Four calcium channel blockers, i.e. nifedipine, verapamil, cinnarizine and tetrandrine are currently available and used widely in treating cardiovascular diseases. To confirm the effects, if any, of calcium channel blockers on cirrhotic patients with portal hypertension, a study was performed on
[Use of calcium-channel blockers in cirrhotic patients with portal hypertension].
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Four calcium-channel blockers (CCB)-nifedipine (NIF), verapamil (VER), cinnarizine (CIN) and tetrandrine (TET)-are currently available and widely used at home and abroad for the treatment of cardiovascular diseases. To confirm any effects of CCBs on cirrhotic patients with portal hypertension, the
Ebola virus. Two-pore channels control Ebola virus host cell entry and are drug targets for disease treatment.
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Ebola virus causes sporadic outbreaks of lethal hemorrhagic fever in humans, but there is no currently approved therapy. Cells take up Ebola virus by macropinocytosis, followed by trafficking through endosomal vesicles. However, few factors controlling endosomal virus movement are known. Here we
Combinatorial screening of a panel of FDA-approved drugs identifies several candidates with anti-Ebola activities.
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Ebola virus (EBOV), pathogen of Ebola hemorrhagic fever (EHF), is an enveloped filamental RNA virus. Recently, the EHF crisis occurred in the Democratic Republic of the Congo again highlights the urgency for its clinical treatments. However, no Food and Drug Administration (FDA)-approved
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