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tryptophane/edema
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[Congestive heart failure due to chronic alcoholism (author's transl)].
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In rare instances, chronic alcoholism leads to the congestive heart failure which is characteristic of alcoholic beriberi with edema. The affected patients is usually a young man with longstanding alcoholic intoxication and often with neurologic features. Onset is often sudden, with dyspnea on
Electron microscopic stereology of capillary endothelial cells and cardiomyocytes in artificially arrested canine hearts.
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In open heart surgery and transplantation, sufficient structural preservation of the myocardium immediately following cardioplegic arrest is a precondition for overcoming ischemia and for resumption of postischemic function. Therefore, we compared the protective effect of three clinically applied
Morphometric evaluation of volume shifts between intra- and extra-cellular space before and during global ischemia.
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BACKGROUND
It is well known that all forms of cardiac arrest lead to global ischemia combined with alterations in cellular and interstitial volume. The aim of this study was to investigate the nature of these alterations with respect to different methods of cardiac arrest and establish the extent of
Intraluminal polyethylene glycol stabilizes tight junctions and improves intestinal preservation in the rat.
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Rapidly progressing mucosal breakdown limits the intestinal preservation time below 10 h. Recent studies indicate that intraluminal solutions containing polyethylene glycol (PEG) alleviate preservation injury of intestines stored in UW-Viaspan. We investigated whether a low-sodium PEG solution is
Improved renal function of warm ischemically damaged kidneys using Polysol.
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OBJECTIVE
We sought to assess the efficacy of POLYSOL, a low-viscosity, colloid-based organ preservation solution, for the preservation of warm ischemically damaged kidney grafts compared with histidine-tryptophane-ketoglutarate (HTK) solution.
METHODS
Pigs (25-30 kg) underwent a left nephrectomy
Attenuation of microvascular reperfusion injury in rat pancreas transplantation by L-arginine.
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BACKGROUND
Despite improving results, exocrine complications remain a major challenge in clinical pancreas transplantation. The etiology of posttransplantation pancreatitis relates almost certainly to cold ischemia/reperfusion-induced microvascular injury with an imbalance of vasoconstricting and
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