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tyramine/hemorragia
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Página 1 a partir de 20 resultados
EFFECT OF HEMORRHAGIC SHOCK ON RELEASE OF NOREPINEPHRINE BY TYRAMINE.
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[Intracranial hemorrhage following use of MAO inhibitor tranylcypromine and beer].
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A case is reported of non-fatal acute cerebral haemorrhage in a 47-year-old female who was taking tranylcypromine and who drank 500 ml of normal beer. Since the tyramine contents of several beers, including low alcohol and alcohol free beer, are similar, it is recommended that patients taking
Vascular smooth muscle desensitization in rabbit epigastric and mesenteric arteries during hemorrhagic shock.
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The decompensatory phase of hemorrhage (shock) is caused by a poorly defined phenomenon termed vascular hyporeactivity (VHR). VHR may reflect an acute in vivo imbalance in levels of contractile and relaxant stimuli favoring net vascular smooth muscle (VSM) relaxation. Alternatively, VHR may be
Intravenous 6-hydroxydopamine attenuates vasopressin and oxytocin secretion stimulated by hemorrhage and hypotension but not hyperosmolality in rats.
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The present study sought to determine whether chemical destruction of peripheral catecholaminergic fibers with 6-hydroxydopamine (6OHDA) attenuates vasopressin (VP) and oxytocin (OT) secretion stimulated by hemorrhage, hypotension, and hyperosmolality. Rats received 6OHDA (100 mg/kg iv) or vehicle
Modulation of jejunal ion and water absorption by endogenous angiotensin after hemorrhage.
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In the pentobarbital sodium-anesthetized rat, hemorrhage of blood equivalent to 1% body weight (18.4% blood volume) increases plasma renin activity and plasma aldosterone concentration and also markedly elevates jejunal ion and water absorption. Infusion of angiotensin II (AII) also stimulates
Effect of subarachnoid hemorrhage on serotonin uptake and release in the rabbit basilar artery.
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This study analyzes the changes induced by subarachnoid hemorrhage (SAH) on the serotonin (5-hydroxytryptamine, 5-HT) uptake and release evoked in rabbit basilar arteries by tyramine. Rabbits were injected with 5 ml of autologous arterial blood into the cisterna magna to produce SAH. Tritium
Constriction of canine prenodal lymphatic vessels following the intra-arterial injection of vasoactive agents and hemorrhage.
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In the forelimbs of anesthetized dogs, perfused at constant arterial inflow, we measured the pressure in a prenodal lymphatic vessel before and following arterial hemorrhage to a mean systemic arterial pressure of approximately 55 mmHg. We also made bolus intra-arterial injections of 1 microgram
Dietary tyramine and other pressor amines in MAOI regimens: a review.
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A critical review of the literature on amine composition and relevant case reports provides rational guidelines for diet planning and counseling of patients on monoamine oxidase inhibitor (MAOI) drug regimens. Small amounts of normally harmless pressor amines in foods can lead to a hypertensive
HYPOTENSION AND LOSS OF PRESSOR RESPONSE TO ANGIOTONIN AS THE RESULT OF TRAUMA TO THE CENTRAL NERVOUS SYSTEM AND SEVERE HEMORRHAGE.
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1. Angiotonin refractoriness and hypotension follow upon injury to the central nervous system in dogs and cats. Refractoriness does not develop when the nervous system is quickly and expertly destroyed or the activity of the nervous system depressed by widespread injection into it of a local
The monoamine oxidase inhibitor-tyramine interaction.
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Reports of hypertensive reactions from monoamine oxidase inhibitors (MAOI) began to proliferate in the early 1960s. Asatoor did extensive research and found that the combination of an MAOI and a food containing tyramine resulted in the hypertensive interaction ("the cheese reaction"). Because of the
The assessment of potential drug interactions with a new tricyclic antidepressant drug.
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1 Methods for the investigation of possible interactions with tricyclic antidepressant drugs are described. These methods have been applied to a new compound, Ciba 34276-Ba, which has been shown to have antidepressant activity. 2 In five normal volunteers tested before and during treatment with Ciba
Alterations in reflex function contributing to syncope: orthostatic hypotension, carotid sinus hypersensitivity and drug-induced dysfunction.
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Orthostatic hypotension and related neurologic symptoms are frequently encountered in clinical practice. The maintenance of appropriate blood pressure and heart rate responses upon assuming the upright posture are dependent upon: 1. intact mechanical (venous valves) mechanisms, 2. functioning
Cardiovascular responsiveness to vasoactive agents in rats with obstructive jaundice.
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1. Pressor responses to tyramine, beta-phenylethylamine and angiotensin II were normal in urethane-anaesthetized rats 7 days after division of the common bile duct. The pressor response to noradrenaline was enhanced at the highest dose level used. 2. The hypotensive response to haemorrhage was
Selective chemical hepatic sympathectomy in the dog.
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Intraportal injection of 6-hydroxydopamine (6-OHDA) was used to produce selective hepatic sympathectomy in the dog. Previously reported techniques for 6-OHDA induced hepatic sympathectomy in rats and cats were modified considerably using alpha and beta adrenergic blocking agents to prevent the
The effect of bile duct manipulation on cardiovascular responsiveness: in vivo studies.
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Cholestasis depresses cardiovascular function and responsiveness. In a previous study, the 3-day bile duct manipulated (BDM) rat was validated as the appropriate control for studying in vitro vascular neuroeffector mechanisms in cholestasis. The present study reports the findings on the effect of
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