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wilms tumor/carbohydrate
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Studies of hypertension and carbohydrate intolerance associated with Wilms tumor.
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Carcinoembryonic proteins produced by Wilms' tumor cells in vitro and in vivo.
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A Wilms' tumor cell line (HFWT) was established after tissue culture of the Wilms' tumor which had developed in the left kidney of a five-month-old boy. The HFWT line has the following cyto-biological characteristics: 1. The cells have a spindle, round or polygonal shape with neoplastic and
Lectin histochemistry of nephroblastoma (Wilms' tumour).
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Paraffin sections of seven cases of nephroblastoma and one case of clear cell sarcoma were stained with a battery of eleven lectins conjugated to horseradish peroxidase. Lectin staining revealed similarities between blastema and stroma with respect to their content of glycoconjugates whereas
Energy metabolism in neuroblastoma and Wilms tumor.
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To support high proliferation, the majority of cancer cells undergo fundamental metabolic changes such as increasing their glucose uptake and shifting to glycolysis for ATP production at the expense of far more efficient mitochondrial energy production by oxidative phosphorylation (OXPHOS), which at
The expression of 3-fucosylated-N-acetyl lactosamine carbohydrate determinants in renal tumours.
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The distribution in renal tumours of 3-fucosyl-N-acetyl lactosamine has been studied by using the monoclonal antibodies AGF 4.36 and AGF 4.48 and immunoperoxidase methods on tissue sections. Seven of 19 nephroblastomas and 12 of 30 renal cell carcinomas contained the epitope. In nephroblastomas the
[Hormonal and metabolic criteria of the adequacy of anesthesia during surgery of nephroblastoma in children].
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The data are presented on changes in hypophyseal-adrenal system and carbohydrate metabolism during transperitoneal nephrectomy in 42 children aged 1 to 15 years with stage II-III tumors. In 22 patients surgery was performed under combined neuroleptanalgesia with halothane for induction and
Survivin selective inhibitor YM155 induce apoptosis in SK-NEP-1 Wilms tumor cells.
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BACKGROUND
Survivin, a member of the family of inhibitor of apoptosis proteins, functions as a key regulator of mitosis and programmed cell death. YM155, a novel molecular targeted agent, suppresses survivin, which is overexpressed in many tumor types. The aim of this study was to determine the
[Lectin histochemistry in the kidney and renal tumors].
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In recent years histochemical methods using lectins with diverse carbohydrate binding specificities have proved useful tools for studying the distribution pattern of intracellular and pericellular glycoconjugates at the tissue level. Several studies of human kidneys have shown that the binding sites
Sulfated HNK-1 epitope in developing and mature kidney: a new marker for thin ascending loop of Henle and tubular injury in acute tubular necrosis.
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The HNK-1 carbohydrate epitope is a 3-sulfo-glucuronyl residue attached to lactosamine structures on glycoproteins, proteoglycans, or glycolipids mostly expressed in the nervous system. Here, using monoclonal antibodies against the sulfated HNK-1 carbohydrate epitope, we first examined its
Expression of polysialic acid and STX, a human polysialyltransferase, is correlated with tumor progression in non-small cell lung cancer.
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Polysialic acid (PSA) is a carbohydrate composed of a linear homopolymer of alpha-2-8-linked sialic acid residues and is mainly attached to the neural cell adhesion molecule (NCAM). Because of the large negative charge of PSA, presence of PSA attenuates the adhesive property of NCAM and increases
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