asphyxia/seizures

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[Metabolic kinetics in the brains in infants with IUGR, respiratory distress syndrome, seizures and asphyxia].

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31-P magnetic resonance spectroscopy (MRS) allows noninvasive measurements of cerebral phosphorus compounds: ATP, phosphocreatine (PCr), inorganic phosphate (Pi), phosphomonoesters (PME) and phosphodiesters (PDE). In this paper we reported our MRS data from the brains of infants with intrauterine

Abusive suffocation presenting as new-onset seizure.

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Child abuse can often be very difficult to identify. This is especially true in cases of abuse by suffocation. Suffocation often leaves no external physical marks and presents with vague, nonspecific symptoms. Infants who have been suffocated usually present unexplained apnea, cyanosis, or seizure.

Pre-existing hypoxia is associated with greater EEG suppression and early onset of evolving seizure activity during brief repeated asphyxia in near-term fetal sheep.

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Spontaneous antenatal hypoxia is associated with high risk of adverse outcomes, however, there is little information on neural adaptation to labor-like insults. Chronically instrumented near-term sheep fetuses (125 ± 3 days, mean ± SEM) with baseline PaO2 < 17 mmHg (hypoxic group: n = 8) or > 17

nNOS inhibition during profound asphyxia reduces seizure burden and improves survival of striatal phenotypic neurons in preterm fetal sheep.

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Basal ganglia injury after hypoxia-ischemia remains common in preterm infants, and is closely associated with later cerebral palsy. In the present study we tested the hypothesis that a highly selective neuronal nitric oxide synthase (nNOS) inhibitor, JI-10, would improve survival of striatal

[Preventive treatment of convulsions in perinatal asphyxia].

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A double blind randomized study has been performed in 17 newborn infants bearing a diagnosis of perinatal asphyxia and treated with phenobarbital (FB) or phenytoin (DPH) to prevent the onset of seizures. The initial dose for both drugs was 12 m/kg IM the first day, followed by 6 mg/kg/day through

Predictors of outcome in term infants with neonatal seizures subsequent to intrapartum asphyxia.

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The objective of this study was to define potential clinical prognostic factors for term infants with neonatal seizures subsequent to intrapartum asphyxia. The authors completed a retrospective analysis of 62 term infants with clinical neonatal seizures subsequent to intrapartum asphyxia. Logistic

Seizure-associated brain injury in term newborns with perinatal asphyxia.

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BACKGROUND There is controversy over whether seizures, the most common manifestation of neonatal brain injury, may themselves damage the developing brain. OBJECTIVE To determine if neonatal seizures are independently associated with brain injury in newborns with perinatal asphyxia. METHODS Ninety

OXYTOCIN REDUCES SEIZURE BURDEN AND HIPPOCAMPAL INJURY IN A RAT MODEL OF PERINATAL ASPHYXIA.

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Foetal asphyxia, a frequent birth complication, detrimentally impacts the immature brain, resulting in neuronal damage, uncontrolled seizure activity and long-term neurological deficits. Oxytocin, a neurohormone mediating important materno-foetal interactions and parturition, has been

Brain alkalosis causes birth asphyxia seizures, suggesting therapeutic strategy.

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OBJECTIVE The mechanisms whereby birth asphyxia leads to generation of seizures remain unidentified. To study the possible role of brain pH changes, we used a rodent model that mimics the alterations in systemic CO(2) and O(2) levels during and after intrapartum birth asphyxia. METHODS Neonatal rat

Acid extrusion via blood-brain barrier causes brain alkalosis and seizures after neonatal asphyxia.

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Birth asphyxia is often associated with a high seizure burden that is predictive of poor neurodevelopmental outcome. The mechanisms underlying birth asphyxia seizures are unknown. Using an animal model of birth asphyxia based on 6-day-old rat pups, we have recently shown that the seizure burden is

III. A Case of Stasis Cyanosis following an Epileptic Seizure, Simulating Traumatic Asphyxia.

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Seizure-associated brain injury in term newborns with perinatal asphyxia.

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Choking, asphyxiation and the insular seizure.

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The insular cortex is located deep within the Sylvian fissure, and has rich connections. We describe two patients with focal epilepsy arising from this area, with symptoms of choking and strangulation during consciousness. Clinicians should be aware of this unusual presentation and that interictal

Neonatal Lethargy, Seizures, and Asphyxiation.

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[Fatal case of asphyxia in asthmatic seizure].

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