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lysozyme/hemoragie

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The pathogenesis of gastric antral hemorrhage and ulceration is unclear. This paper first proposes that antral hemorrhagic ulcers produced in rats are associated with attenuation of defensive parameters (such as mucosal glutathione levels and histamine release, as well as aggravation of aggressive
The pathogenic mechanisms underlying betel quid chewing (BQC)-induced gastric haemorrhagic ulcer are totally unknown. This study first demonstrated that BQC produced gastric haemorrhagic ulcer via oxidative stress that could be protected by lysozyme chloride and glutathione in rats. Male Wistar rats

Effect of lysozyme chloride on betel quid chewing aggravated gastric oxidative stress and hemorrhagic ulcer in diabetic rats.

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OBJECTIVE To evaluate the protective effect of lysozyme chloride on betel quid chewing (BQC) aggravated gastric oxidative stress and hemorrhagic ulcer in rats with diabetes mellitus (DM). METHODS Male Wistar rats were challenged intravenously with streptozotocin (65 mg/kg) to induce DM. Rats were

[Correlation of immunomodulating effects induced by lysozyme and naphthoquinones in acute blood loss].

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The role of erythrocytes in realization of interrelation of the immunomodulating effects of lysozyme and naphthoquinones in normal and under conditions of acute hemorrhage was studied. Interaction of lysozyme and menadione at the level of intact erythrocytes and their stroma resulted in formation of

[Changes in serum lysozyme activity in terminal states caused by blood loss].

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A study was made of the dynamics of the lysozyme activity of the serum in dogs which sustained clinical death on the background of a 2-hour hypovolemic hypotension due to blood loss. There was revealed a progressive increase in the lysozyme activity of the serum during the hypotension and during the

[Effect of lysozyme on the functional and metabolic activity of polymorphonuclear leukocytes in the acute blood loss].

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Acute hemorrhage leads to a decrease in the phagocyte number and index, the NBT test parameters, and the NADPH oxidase activity and to an increase in the Ca2+ ATPase activity in polymorphonuclear leukocytes on the peripheral blood. Lysozyme produces effective correction of the above parameters of

Protective effect of lysozyme chloride on gastric oxidative stress and hemorrhagic ulcers in severe atherosclerotic rats.

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BACKGROUND Calcium and cholesterol play major roles in the formation of atherosclerosis. Whether severe atherosclerosis induced by co-administration of a mixture containing vitamin D2 (vit D2) and cholesterol can result in gastric hemorrhagic damage is unknown. Gastric oxidative stress and

Dextran-Conjugated Lysozymes Inhibit the Growth of Shigella sonnei and Viral Hemorrhagic Septicemia Virus.

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Lysozyme is well known as a natural antimicrobial agent, but its function is limited in that it only combats Gram-positive bacteria. We investigated the inhibitory effects of dextran-conjugated lysozymes (DLs) against some strains of Gram-negative bacteria and viral hemorrhagic septicemia virus

[Immuno-metabolic effects of lysozyme in acute blood loss].

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Lysozyme injections (1 or 5 mg/kg) produced marked immunomodulating, antioxidant and hepatoprotective effects in Wistar rats which had lost much blood (3% body mass). Lysozyme action is mediated by heavy erythrocytes and glass-adherent splenocytic cytokines the production of which is provoked by

Renal damage in hemorrhagic fever with renal syndrome. Measurements of urine and serum lysozyme and urine IgG.

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[Lysozyme in association with vitamins C and K in the treatment of some hemorrhagic conditions in surgery].

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[Utilization in ORL of the anti-hemorrhagic properties of lysozyme].

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[Behavior of the blood lysozyme level after acute hemorrhage in rabbits].

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OBJECTIVE Our recent studies proved that menstrual discharge is exceptionally rich in bactericidal hemoglobin peptides (hemocidins). Of special interest is the behavior of hemocidins in low pH of the vagina, in different ionic strengths, and in the presence of other specialized antibacterial

Hemorrhagic intracranial inflammatory pseudotumor originating from the trigeminal nerve: a case report.

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We report here on a case of intracranial inflammatory pseudotumor arising from the trigeminal nerve. A 52-year-old man presented with sudden onset severe headache. He had had facial numbness several months earlier and no signs indicating infection. On the computerized tomography scan, intracranial
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