polycythemia/nicotine

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Tobacco smoking causes secondary polycythemia and a mild leukocytosis among heavy smokers in Taif City in Saudi Arabia.

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Tobacco smoking is a common risk factor of cardiovascular diseases, cancers and heart health problems. In Taif, the number of secondary polycythemia patients is increasing dramatically and most of those patients are heavy smokers. Therefore, this study is an attempt to understand the

[Acute myocardial infarct caused by nicotine-induced erythrocytosis].

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A 29-year-old heavy smoker presented with an acute myocardial infarction and hematocrit of 70%. At immediate coronary angiography a complete occlusion of the right coronary artery was found. After intracoronary urokinase the coronary arteries were found to be completely normal. Causes for the

Polycythemia and Anemia in Hereditary Hemochromatosis.

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Introduction Hereditary hemochromatosis is a syndrome of dysregulated iron homeostasis resulting in the excessive deposition of iron. Hemochromatosis causes pulmonary, pancreatic, and hepatic dysfunction, all of which are risk factors for anemia in the general population. Conversely, iron overload

[Relationship between tobacco smoke exposure and the concentrations of carboxyhemoglobin and hemoglobin].

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To determine the carboxyhemoglobin (CO-Hb) predictive intervals in active and passive smokers and to obtain an equation expressing the relation of CO-Hb to number of cigarettes smoked, we studied 233 outpatients referred to an urban university hospital for arterial gas measurement. Patients were

[Tobacco consumption and carboxyhemoglobin levels in blood donors].

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The authors studied the tobacco consumption of 283 blood donors and its consequences. 151 were nonsmokers and 132 were smokers (47.7% smoke more than 10 g/day). Their mean HbCO level was 4.3 +/- 0.2% (highest level 15%). HbCO levels are significantly correlated with daily tobacco consumption (p less

Acute and Chronic Carbon Monoxide Toxicity from Tobacco Smoking.

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Carbon monoxide (CO) is produced from incomplete combustion of hydrocarbons and is a by-product of tobacco smoking. Chronic cigarette smokers often have carboxyhemoglobin (COHb) concentrations as high as 10%. We report a case of severely elevated COHb and polycythemia because of

[Differential Diagnosis of Erythrocytosis - Background and Clinical Relevance].

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Due to its rare incidence, erythrocytosis frequently represents a challenge for the treating doctors. The erythropoiesis (= production of erythrocytes) is located in the bone marrow, and the hormone erythropoietin (EPO) takes control in its regulation. Therefore, measurement of EPO in serum is one

Gaisböck syndrome (polycythemia and hypertension) revisited: results from the national inpatient sample database.

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BACKGROUND Polycythemia is characterized by increased blood viscosity and a chronic inflammatory state possibly giving rise to excessive thromboembolic events and hypertensive cardiovascular disease. We aimed to study the relationship between polycythemia and cardiac risk factors using a large

An unhealthy blush - secondary erythrocytosis due to waterpipe smoking

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Introduction: We present a case of a patient with a chronic carbon monoxide (CO) intoxication with facial plethora due to secondary erythrocytosis. Case details: A 22-year-old

Association between cigarette & shisha smoking and the severity of polycythemia: A cross sectional study.

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Tobacco cigarette smoking is assumed to be a key reason of death all over the world. Smoking had both severe as well long-lasting effects on hematological constraints. As per the data available from World Health Organization, every year nearly 5 million individuals die around the world due to the

[The polycythemias].

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The primary polycythemias result from malignant proliferation of myeloid stem-cell. Typically, an increase of red cell mass and a decrease of erythropoietin is found. In polycythemia vera, augmentation of PCV is frequently associated with elevation of WBC and platelets, as well as splenomegaly. The

Polycythemia vera and other polycythemic states.

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The diagnosis of polycythemia requires an accurate and independent assessment of both plasma volume and red blood cell mass. Patients with an increased red cell mass (absolute polycythemia) may be hypoxic or have an erythropoietin-secreting tumor or space-occupying lesion compressing the kidney.

Does untreated obstructive sleep apnea cause secondary erythrocytosis?

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BACKGROUND The current literature suggests a relationship between obstructive sleep apnea (OSA) severity and hematocrit. However, the degree that OSA contributes to clinically significant erythrocytosis is uncertain. The aim of this study is to evaluate this association in a large study sample

Thromboembolic complications of polycythemia: polycythemia vera versus smokers' polycythemia.

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OBJECTIVE This report compares patients with the hypercoagulable state of polycythemia vera to patients with secondary polycythemia caused by tobacco use to determine whether the incidence of thromboembolic events is equivalent. METHODS The medical records of 146 patients with the diagnosis of

[Secondary erythrocytoses].

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The author provides data pertinent to the modern classification of erythrocytosis, which rests on the pathogenetic principle. The difference in the pathogenesis of erythremia and secondary erythrocytoses was proved with the help of an erythroid culture and by examination of erythropoietins, which

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