potassium chloride/infarction

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[The hemorrhagic infarction of the cecum. A complication after the intake of potassium-chloride tablets (author's transl)].

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Case report of a 28 year old female patient who had a localized gangrene of the cecum after the intake of Kalinor-Dragées an enteric-coated potassium tablet. It is emphasized that the enteric-coated potassium-tablets have a higher complication rate of stenosis, ulceration and spontaneous perforation

[EFFECTS OF THE INFUSION OF A SOLUTION OF GLUCOSE, INSULIN AND POTASSIUM CHLORIDE ON THE CLINICAL AND ELECTROCARDIOGRAPHICAL EVOLUTION OF MYOCARDIAL INFARCT IN THE AGED].

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Effects of intracoronary potassium chloride on electrograms of canine Purkinje fibers in six-hour- to four-week-old myocardial infarcts. An indication of time-dependent changes in collateral blood flow.

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[The treatment of patients with myocardial infarct with potassium chloride and polarizing solution].

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[Effect of nezaton and potassium chloride on the animal's sensitivity to strophanthin in experimental myocardial infarct].

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[Use of potassium chloride in myocardial infarction].

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Comparative effect of 15 per cent potassium chloride and 30 per cent potassium citrate in resuscitation from ventricular fibrillation following acute myocardial infarction; an experimental study.

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AR-R15896AR reduces cerebral infarction volumes after focal ischemia in cats.

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OBJECTIVE The use of competitive and noncompetitive N-methyl-D-aspartate receptor antagonists to prevent neuronal death during ischemia has been comprehensively studied. This study was performed to examine the neuroprotective effects and pharmacokinetics of the noncompetitive N-methyl-D-aspartate

Infarct tolerance against temporary focal ischemia following spreading depression in rat brain.

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A rat model of ischemic tolerance is useful for studying the intrinsic cellular mechanism of resistance to cerebral ischemia. Many types of preconditioning in the brain have been reported to induce ischemic tolerance; however, evaluation of their neuroprotective effect is primarily limited to

Does magnesium modify left ventricular remodeling after experimental myocardial infarction?

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BACKGROUND Magnesium therapy has been shown to improve survival and decrease the incidence of left ventricular failure when given to patients shortly after a myocardial infarction, but the mechanisms are unknown. We tested the hypothesis that the benefits of magnesium therapy are due to a favorable

[Metabolic effects of a glucose-insulin-potassium mixture in acute myocardial infarct].

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In order to limit the spread of myocardial infarction, 56 patients with acute myocardial infarction, admitted to hospital within 10 hours from the attack, were for two days treated with continuous intravenous drip infusion of glucose-insulin-potassium mixture (GIP) (300 ml glucose, 50 U common

Effects of glucose-insulin-potassium infusion on myocardial perfusion and left ventricular remodeling in patients treated with primary angioplasty for ST-elevation acute myocardial infarction.

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The role of glucose-insulin-potassium (GIK) infusion in the management of acute coronary syndrome is controversial. Limited data are available on the effects of adjunctive high-dose GIK (30% glucose, 50 IU of insulin, 80 mEq of potassium chloride infused at 1.5 ml/kg/hour over 24 hours) on

Effects of glucose-insulin-potassium infusion on ST-elevation myocardial infarction in patients treated with thrombolytic therapy.

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The role of glucose-insulin-potassium (GIK) infusion in the management of acute myocardial infarction is not well established. This prospective, randomized study comprised 120 patients who had ST-elevation myocardial infarction that was treated within 12 hours from symptom onset with a high dose of

Evaluation of exercise and potassium chloride supplementation on blood pressure and nociceptive threshold in hypertensive rats.

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Hypertensive subjects present an increased nociceptive threshold, and the lack or delay of pain perception may impede detection of angina and myocardial infarction. Nutritional interventions, like potassium chloride (KCl) diet supplementation, and exercises are common nonpharmacological indications

Effects of glucose-insulin-potassium solution added to reperfusion treatment in acute myocardial infarction.

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OBJECTIVE Reperfusion treatment modalities used in the routine treatment protocols of acute myocardial infarction (AMI) were found to be ineffective in establishing the nutritional cellular reperfusion in the microvascular environment even they succeed to open the infarct related artery.

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