salicylic acid/hemoragie

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[Behaviour of "haemorrhage times" in hypocolinesterasic subjects after administration of acetil-salicylic acid (author's transl)].

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The competitive action of acetil-salicylic acid with respect to colinesterasi is demonstrated. The A. proposes a routine dose of this enzyme in the preparation for surgery with a high haemorrhage risk, such as tonsillectomy.

Interaction of salicylic acid with adenosine and adenosine triphosphate. Potential mechanism of intensifying acetylsalicylic acid-induced GI blood loss.

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Complex formation between salicylic acid and adenosine or adenosine triphosphate in 0.2m phosphate buffer at pH=7 was investigated as a potential factor contributing to the prolongation of acetylsalicylic acid-induced GI blood loss. Spectrophotemetric techniques were used to evalute the

Plasma levels of acetylsalicylic acid and salicylic acid after oral ingestion of plain and buffered acetylsalicylic acid in relation to bleeding time and thrombocyte function.

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Buffered acetylsalicylic acid (Alka Seltzer, B-ASA) and plain aspirin (P-ASA) tablets were compared as to their effects on bleeding time and platelet function in eight healthy male volunteers. Two doses (500 and 1000 mg) of each preparation were investigated in a cross-over design, each volunteer

Interactions between sodium salicylate and acetyl salicylic acid evaluated using ADP induced platelet aggregation and bleeding time.

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1 g acetyl salicylic acid orally significantly enhanced the initial rate of platelet aggregation induced by 1 mumol/l and 2.5 mumol/l ADP. Sodium salicylate was without effects on the platelet aggregation and specifically it did not prevent acetylsalicylic acid from inhibiting the secondary

The interaction of varying doses of dipyridamole and acetyl salicylic acid on the inhibition of platelet functions and their effect on bleeding time.

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1 In normal volunteers maximum reductions in platelet functions, collagen aggregation, adhesion and PF4 availability, were achieved using combined doses of 50 mg three times daily dipyridamole + 180 mg ASA or 75 mg three times daily dipyridamole + 120 mg ASA daily. 2 These doses did not prolong the

The effect of DDAVP and placebo on platelet function and prolonged bleeding time induced by oral acetyl salicylic acid intake in healthy volunteers.

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[Comparative randomized open study of the efficacy and tolerance of enemas with 2 gr of 4-amino-salicylic acid (4-ASA) and 1 gr of 5-amino-salicylic acid (5-ASA) in distal forms of hemorrhagic rectocolitis].

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OBJECTIVE The aim of this multicentre, randomized open trial was to compare the efficacy and tolerance of 4-ASA vs 5-ASA enemas in the treatment of distal moderately active ulcerative colitis. Fifty patients were randomized to receive enemas in 100 mL suspension of 4-ASA (2 g, n = 26), or 5-ASA (1

[Variation of bleeding time after administration of acetyl salicylic acid at different doses in the healthy volunteer].

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Template bleeding time after ingestion of ultra low dosages of acetyl salicylic acid in healthy subjects. Preliminary study.

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[Recurrent gastric hemorrhage caused by salicylic acid].

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[Salicylic acid poisoning as a cause of severe hemorrhagic diathesis].

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[Letter: Gastric hemorrhage caused by salicylic acid and its active diagnosis].

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[Gastrointestinal hemorrhage after salicylic acid therapy in peptic ulcer of long duration].

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Interaction of salicylic acid with adenosine and adenosine triphosphate: potential mechanism of intensifying aspirin-induced GI blood loss.

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[Fatal bleeding under warfarin plus cranberry juice. Is it due to salicylic acid?].

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